Pages that link to "Q28363890"
Jump to navigation
Jump to search
The following pages link to Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death (Q28363890):
Displaying 50 items.
- Titanium dioxide (TiO2) nanoparticles preferentially induce cell death in transformed cells in a Bak/Bax-independent fashion (Q21133813) (← links)
- Regulation of the mitochondrial apoptosis-induced channel, MAC, by BCL-2 family proteins (Q21564051) (← links)
- Hypoxia and defective apoptosis drive genomic instability and tumorigenesis (Q22242660) (← links)
- Tumor necrosis factor-alpha induces Bax-Bak interaction and apoptosis, which is inhibited by adenovirus E1B 19K (Q24291723) (← links)
- Bcl-x(L) sequesters its C-terminal membrane anchor in soluble, cytosolic homodimers (Q24292693) (← links)
- A novel 21-kDa cytochrome c-releasing factor is generated upon treatment of human leukemia U937 cells with geranylgeraniol (Q24293188) (← links)
- Active Bax and Bak are functional holins (Q24293539) (← links)
- FLASH links the CD95 signaling pathway to the cell nucleus and nuclear bodies (Q24294356) (← links)
- tBid undergoes multiple conformational changes at the membrane required for Bax activation (Q24294675) (← links)
- The Molecular Mechanism of Noxa-induced Mitochondrial Dysfunction in p53-Mediated Cell Death (Q24294863) (← links)
- BAK multimerization for apoptosis, but not bid binding, is inhibited by negatively charged residue in the BAK hydrophobic groove (Q24295530) (← links)
- Noxa is necessary for hydrogen peroxide-induced caspase-dependent cell death (Q24296352) (← links)
- Identification of novel isoforms of the BH3 domain protein Bim which directly activate Bax to trigger apoptosis (Q24296925) (← links)
- Inhibition of Bid-induced apoptosis by Bcl-2. tBid insertion, Bax translocation, and Bax/Bak oligomerization suppressed (Q24297109) (← links)
- PKC-ι promotes glioblastoma cell survival by phosphorylating and inhibiting BAD through a phosphatidylinositol 3-kinase pathway (Q24297584) (← links)
- Molecular cloning and characterization of a human AIF-like gene with ability to induce apoptosis (Q24297933) (← links)
- Proapoptotic BAX and BAK control multiple initiator caspases (Q24298402) (← links)
- Bcl-x(L) retrotranslocates Bax from the mitochondria into the cytosol (Q24298758) (← links)
- High-throughput methods to detect dimerization of Bcl-2 family proteins (Q24299128) (← links)
- An endoplasmic reticulum stress-specific caspase cascade in apoptosis. Cytochrome c-independent activation of caspase-9 by caspase-12 (Q24300914) (← links)
- Humanin peptide suppresses apoptosis by interfering with Bax activation (Q24301403) (← links)
- Prevention of cytokine withdrawal-induced apoptosis by Mcl-1 requires interaction between Mcl-1 and Bim (Q24301935) (← links)
- Roles of CHOP/GADD153 in endoplasmic reticulum stress (Q24302185) (← links)
- Interaction of F1L with the BH3 domain of Bak is responsible for inhibiting vaccinia-induced apoptosis (Q24302248) (← links)
- Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins (Q24302494) (← links)
- Functional and physical interaction between Bcl-X(L) and a BH3-like domain in Beclin-1 (Q24303558) (← links)
- IBRDC2, an IBR-type E3 ubiquitin ligase, is a regulatory factor for Bax and apoptosis activation (Q24303971) (← links)
- UV irradiation resistance-associated gene suppresses apoptosis by interfering with BAX activation (Q24304043) (← links)
- Heterodimerization of BAK and MCL-1 activated by detergent micelles (Q24305060) (← links)
- BH3 domains other than Bim and Bid can directly activate Bax/Bak (Q24305138) (← links)
- ASC is a Bax adaptor and regulates the p53-Bax mitochondrial apoptosis pathway (Q24305337) (← links)
- Differential contribution of Puma and Noxa in dual regulation of p53-mediated apoptotic pathways (Q24306130) (← links)
- Bifunctional Apoptosis Regulator (BAR), an Endoplasmic Reticulum (ER)-associated E3 Ubiquitin Ligase, Modulates BI-1 Protein Stability and Function in ER Stress (Q24306658) (← links)
- hsp70-DnaJ chaperone pair prevents nitric oxide- and CHOP-induced apoptosis by inhibiting translocation of Bax to mitochondria (Q24306768) (← links)
- Human alphaA- and alphaB-crystallins bind to Bax and Bcl-X(S) to sequester their translocation during staurosporine-induced apoptosis (Q24306784) (← links)
- Quantitation of mitochondrial dynamics by photolabeling of individual organelles shows that mitochondrial fusion is blocked during the Bax activation phase of apoptosis (Q24307662) (← links)
- A stapled BID BH3 helix directly binds and activates BAX (Q24307663) (← links)
- Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1 (Q24308005) (← links)
- The RAB25 small GTPase determines aggressiveness of ovarian and breast cancers (Q24308778) (← links)
- GIDE is a mitochondrial E3 ubiquitin ligase that induces apoptosis and slows growth (Q24308802) (← links)
- BimL directly neutralizes Bcl-xL to promote Bax activation during UV-induced apoptosis (Q24309169) (← links)
- TRIM39 is a MOAP-1-binding protein that stabilizes MOAP-1 through inhibition of its poly-ubiquitination process (Q24309591) (← links)
- The vaccinia virus protein F1L interacts with Bim and inhibits activation of the pro-apoptotic protein Bax (Q24310440) (← links)
- Bax crystal structures reveal how BH3 domains activate Bax and nucleate its oligomerization to induce apoptosis (Q24313049) (← links)
- Chemical genomics identifies small-molecule MCL1 repressors and BCL-xL as a predictor of MCL1 dependency (Q24315549) (← links)
- Clusterin inhibits apoptosis by interacting with activated Bax (Q24316499) (← links)
- A functional role for nicotine in Bcl2 phosphorylation and suppression of apoptosis (Q24317110) (← links)
- The tyrosine kinase Lck is a positive regulator of the mitochondrial apoptosis pathway by controlling Bak expression (Q24317331) (← links)
- Physical and functional interaction between BH3-only protein Hrk and mitochondrial pore-forming protein p32 (Q24318108) (← links)
- RACK1 promotes Bax oligomerization and dissociates the interaction of Bax and Bcl-XL (Q24319133) (← links)