Pages that link to "Q28201848"
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The following pages link to Evidence for BLM and Topoisomerase IIIalpha interaction in genomic stability (Q28201848):
Displayed 50 items.
- BLAP75, an essential component of Bloom's syndrome protein complexes that maintain genome integrity (Q24298321) (← links)
- Human topoisomerase IIIalpha is a single-stranded DNA decatenase that is stimulated by BLM and RMI1 (Q24310243) (← links)
- Scaffolding protein SPIDR/KIAA0146 connects the Bloom syndrome helicase with homologous recombination repair (Q24336963) (← links)
- Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome (Q24337192) (← links)
- Recql5 and Blm RecQ DNA helicases have nonredundant roles in suppressing crossovers (Q24522689) (← links)
- Dual localization of human DNA topoisomerase IIIalpha to mitochondria and nucleus (Q24535778) (← links)
- The Bloom's syndrome helicase stimulates the activity of human topoisomerase IIIalpha (Q24537672) (← links)
- MPS1-dependent mitotic BLM phosphorylation is important for chromosome stability (Q24554386) (← links)
- Roles of RECQ helicases in recombination based DNA repair, genomic stability and aging (Q24646853) (← links)
- RMI, a new OB-fold complex essential for Bloom syndrome protein to maintain genome stability (Q24650860) (← links)
- A Multiprotein Nuclear Complex Connects Fanconi Anemia and Bloom Syndrome (Q24669612) (← links)
- Human premature aging, DNA repair and RecQ helicases (Q24680996) (← links)
- Disease-causing missense mutations in human DNA helicase disorders (Q26828898) (← links)
- RecQ helicases in DNA double strand break repair and telomere maintenance (Q27692685) (← links)
- Slx1-Slx4 is a second structure-specific endonuclease functionally redundant with Sgs1-Top3. (Q27932379) (← links)
- Functional overlap between Sgs1-Top3 and the Mms4-Mus81 endonuclease (Q27935913) (← links)
- (Q28158104) (redirect page) (← links)
- Functional interaction of p53 and BLM DNA helicase in apoptosis (Q28201136) (← links)
- The broken genome: genetic and pharmacologic approaches to breaking DNA (Q28299327) (← links)
- The Bloom's syndrome helicase is critical for development and function of the alphabeta T-cell lineage (Q28594855) (← links)
- Srs2 and Sgs1-Top3 suppress crossovers during double-strand break repair in yeast (Q29618612) (← links)
- Promyelocytic leukemia nuclear bodies behave as DNA damage sensors whose response to DNA double-strand breaks is regulated by NBS1 and the kinases ATM, Chk2, and ATR. (Q30480527) (← links)
- SUMO modification regulates BLM and RAD51 interaction at damaged replication forks (Q30940899) (← links)
- Functional relationships of FANCC to homologous recombination, translesion synthesis, and BLM (Q33761695) (← links)
- The HRDC domain of BLM is required for the dissolution of double Holliday junctions (Q33899656) (← links)
- Association of yeast DNA topoisomerase III and Sgs1 DNA helicase: studies of fusion proteins. (Q33944291) (← links)
- Regulation of BLM Nucleolar Localization (Q34047411) (← links)
- Phosphorylation of BLM, dissociation from topoisomerase IIIalpha, and colocalization with gamma-H2AX after topoisomerase I-induced replication damage (Q34097292) (← links)
- Alternative lengthening of telomeres in mammalian cells (Q34114656) (← links)
- The role of post-translational modifications in fine-tuning BLM helicase function during DNA repair (Q34244028) (← links)
- G4 DNA unwinding by BLM and Sgs1p: substrate specificity and substrate-specific inhibition (Q34367524) (← links)
- The dissolution of double Holliday junctions (Q34427421) (← links)
- BLM helicase stimulates the ATPase and chromatin-remodeling activities of RAD54. (Q34659003) (← links)
- Defending genome integrity during S-phase: putative roles for RecQ helicases and topoisomerase III. (Q35037441) (← links)
- Top3α is required during the convergent migration step of double Holliday junction dissolution. (Q35083245) (← links)
- RecQ helicases: suppressors of tumorigenesis and premature aging. (Q35152970) (← links)
- TopBP1 interacts with BLM to maintain genome stability but is dispensable for preventing BLM degradation (Q35218078) (← links)
- Structure and function of RecQ DNA helicases (Q35817571) (← links)
- RMI1 promotes DNA replication fork progression and recovery from replication fork stress (Q36210964) (← links)
- Two closely related RecQ helicases have antagonistic roles in homologous recombination and DNA repair in Arabidopsis thaliana (Q36277053) (← links)
- Absence of BLM leads to accumulation of chromosomal DNA breaks during both unperturbed and disrupted S phases (Q36322513) (← links)
- Bloom's syndrome protein is required for correct relocalization of RAD50/MRE11/NBS1 complex after replication fork arrest (Q36324118) (← links)
- Multimeric BLM is dissociated upon ATP hydrolysis and functions as monomers in resolving DNA structures (Q36341536) (← links)
- Mechanisms of RecQ helicases in pathways of DNA metabolism and maintenance of genomic stability (Q36573932) (← links)
- Unique and important consequences of RECQ1 deficiency in mammalian cells (Q36740600) (← links)
- Collaborating functions of BLM and DNA topoisomerase I in regulating human rDNA transcription (Q36794626) (← links)
- The BLM dissolvasome in DNA replication and repair. (Q37064133) (← links)
- Visualization of human Bloom's syndrome helicase molecules bound to homologous recombination intermediates (Q37327448) (← links)
- Bloom's Syndrome: Clinical Spectrum, Molecular Pathogenesis, and Cancer Predisposition (Q37603027) (← links)
- RecQ helicases: multiple structures for multiple functions? (Q37641761) (← links)