Pages that link to "Q24299852"
Jump to navigation
Jump to search
The following pages link to MDC1 directly binds phosphorylated histone H2AX to regulate cellular responses to DNA double-strand breaks (Q24299852):
Displayed 50 items.
- H2A.X variant histone (Q1620739) (← links)
- MRE11 homolog, double strand break repair nuclease (Q2073928) (← links)
- Nibrin (Q14911994) (← links)
- Double strand breaks can initiate gene silencing and SIRT1-dependent onset of DNA methylation in an exogenous promoter CpG island (Q21092482) (← links)
- Mediator of DNA damage checkpoint 1 (Q21106545) (← links)
- Radiation-induced alterations in histone modification patterns and their potential impact on short-term radiation effects (Q21129305) (← links)
- Phosphorylation: the molecular switch of double-strand break repair (Q21296796) (← links)
- CK2 phosphorylation-dependent interaction between aprataxin and MDC1 in the DNA damage response (Q22001526) (← links)
- Regulation of DNA-damage responses and cell-cycle progression by the chromatin remodelling factor CHD4 (Q24294656) (← links)
- Structural basis of γH2AX recognition by human PTIP BRCT5-BRCT6 domains in the DNA damage response pathway (Q24296259) (← links)
- Wild-type p53-induced phosphatase 1 dephosphorylates histone variant gamma-H2AX and suppresses DNA double strand break repair (Q24297983) (← links)
- The AAA-ATPase VCP/p97 promotes 53BP1 recruitment by removing L3MBTL1 from DNA double-strand breaks (Q24298486) (← links)
- RNF8 ubiquitylates histones at DNA double-strand breaks and promotes assembly of repair proteins (Q24300411) (← links)
- RNF8 transduces the DNA-damage signal via histone ubiquitylation and checkpoint protein assembly (Q24300428) (← links)
- Crystal structure of triple-BRCT-domain of ECT2 and insights into the binding characteristics to CYK-4 (Q24301484) (← links)
- A divalent FHA/BRCT-binding mechanism couples the MRE11-RAD50-NBS1 complex to damaged chromatin (Q24302293) (← links)
- Depletion of nuclear histone H2A variants is associated with chronic DNA damage signaling upon drug-evoked senescence of human somatic cells (Q24305743) (← links)
- RNF8- and RNF168-dependent degradation of KDM4A/JMJD2A triggers 53BP1 recruitment to DNA damage sites (Q24306486) (← links)
- RAP80 targets BRCA1 to specific ubiquitin structures at DNA damage sites (Q24306789) (← links)
- WSTF regulates the H2A.X DNA damage response via a novel tyrosine kinase activity (Q24309061) (← links)
- The Rap80-BRCC36 de-ubiquitinating enzyme complex antagonizes RNF8-Ubc13-dependent ubiquitination events at DNA double strand breaks (Q24309142) (← links)
- The RIDDLE syndrome protein mediates a ubiquitin-dependent signaling cascade at sites of DNA damage (Q24309181) (← links)
- RNF168 binds and amplifies ubiquitin conjugates on damaged chromosomes to allow accumulation of repair proteins (Q24309287) (← links)
- DNA repair. PAXX, a paralog of XRCC4 and XLF, interacts with Ku to promote DNA double-strand break repair (Q24313240) (← links)
- Tyrosine dephosphorylation of H2AX modulates apoptosis and survival decisions (Q24313501) (← links)
- A supramodular FHA/BRCT-repeat architecture mediates Nbs1 adaptor function in response to DNA damage (Q24321669) (← links)
- Vaccinia-related kinase 1 (VRK1) is an upstream nucleosomal kinase required for the assembly of 53BP1 foci in response to ionizing radiation-induced DNA damage (Q24336079) (← links)
- Phospho-dependent interactions between NBS1 and MDC1 mediate chromatin retention of the MRN complex at sites of DNA damage (Q24336328) (← links)
- Phospho-epitope binding by the BRCT domains of hPTIP controls multiple aspects of the cellular response to DNA damage (Q24337134) (← links)
- Rad17 recruits the MRE11-RAD50-NBS1 complex to regulate the cellular response to DNA double-strand breaks (Q24337442) (← links)
- Phosphorylation-dependent PIH1D1 interactions define substrate specificity of the R2TP cochaperone complex (Q24339507) (← links)
- The histone demethylase LSD1/KDM1A promotes the DNA damage response (Q24339556) (← links)
- Epigenetic modifications in double-strand break DNA damage signaling and repair (Q24607287) (← links)
- Dynamics of DNA damage response proteins at DNA breaks: a focus on protein modifications (Q24608343) (← links)
- Mammalian SUMO E3-ligases PIAS1 and PIAS4 promote responses to DNA double-strand breaks (Q24608599) (← links)
- MicroRNA-138 modulates DNA damage response by repressing histone H2AX expression (Q24615514) (← links)
- Molecular insights into the function of RING finger (RNF)-containing proteins hRNF8 and hRNF168 in Ubc13/Mms2-dependent ubiquitylation (Q24633548) (← links)
- Structural genomics of histone tail recognition (Q24634351) (← links)
- Regulation of chromatin by histone modifications (Q24635070) (← links)
- BRCT domains: easy as one, two, three (Q24635835) (← links)
- Structural evidence for direct interactions between the BRCT domains of human BRCA1 and a phospho-peptide from human ACC1 (Q24651980) (← links)
- Orchestration of the DNA-damage response by the RNF8 ubiquitin ligase (Q24653776) (← links)
- Cep164 is a mediator protein required for the maintenance of genomic stability through modulation of MDC1, RPA, and CHK1 (Q24657907) (← links)
- Structural basis for the methylation state-specific recognition of histone H4-K20 by 53BP1 and Crb2 in DNA repair (Q24669691) (← links)
- Ubc13: the Lys63 ubiquitin chain building machine (Q26739716) (← links)
- Writers, Readers, and Erasers of Histone Ubiquitylation in DNA Double-Strand Break Repair (Q26742134) (← links)
- A Novel Aspect of Tumorigenesis-BMI1 Functions in Regulating DNA Damage Response (Q26774980) (← links)
- Functional Role of G9a Histone Methyltransferase in Cancer (Q26781247) (← links)
- Chromatin perturbations during the DNA damage response in higher eukaryotes (Q26782637) (← links)
- Targeting ATM-deficient CLL through interference with DNA repair pathways (Q26822592) (← links)