bcl-Associated Death Protein
"bcl-Associated Death Protein" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A pro-apoptotic protein and member of the Bcl-2 protein family that is regulated by PHOSPHORYLATION. Unphosphorylated Bad protein inhibits the activity of BCL-XL PROTEIN.
| Descriptor ID |
D051025
|
| MeSH Number(s) |
D12.644.360.075.718.100 D12.776.476.075.718.100 D12.776.744.049
|
| Concept/Terms |
bcl-Associated Death Protein- bcl-Associated Death Protein
- Death Protein, bcl-Associated
- bcl Associated Death Protein
- bcl2-Antagonist of Cell Death Protein
- bcl2 Antagonist of Cell Death Protein
- Bad Protein
|
Below are MeSH descriptors whose meaning is more general than "bcl-Associated Death Protein".
Below are MeSH descriptors whose meaning is more specific than "bcl-Associated Death Protein".
This graph shows the total number of publications written about "bcl-Associated Death Protein" by people in this website by year, and whether "bcl-Associated Death Protein" was a major or minor topic of these publications.
View timeline visualization
| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 1 | 1 |
| 2000 | 0 | 1 | 1 |
| 2001 | 0 | 3 | 3 |
| 2002 | 0 | 1 | 1 |
| 2003 | 0 | 3 | 3 |
| 2004 | 0 | 8 | 8 |
| 2005 | 0 | 3 | 3 |
| 2006 | 0 | 3 | 3 |
| 2007 | 0 | 1 | 1 |
| 2008 | 1 | 1 | 2 |
| 2009 | 0 | 2 | 2 |
| 2010 | 0 | 1 | 1 |
| 2011 | 0 | 2 | 2 |
| 2012 | 0 | 1 | 1 |
| 2013 | 0 | 2 | 2 |
| 2014 | 0 | 3 | 3 |
| 2015 | 0 | 2 | 2 |
| 2016 | 0 | 1 | 1 |
| 2017 | 1 | 0 | 1 |
| 2018 | 0 | 1 | 1 |
| 2019 | 0 | 1 | 1 |
Below are the most recent publications written about "bcl-Associated Death Protein" by people in Profiles.
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GPER-induced signaling is essential for the survival of breast cancer stem cells. Int J Cancer. 2020 03 15; 146(6):1674-1685.
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Ubiquinol promotes retinal ganglion cell survival and blocks the apoptotic pathway in ischemic retinal degeneration. Biochem Biophys Res Commun. 2018 09 18; 503(4):2639-2645.
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The RhoJ-BAD signaling network: An Achilles' heel for BRAF mutant melanomas. PLoS Genet. 2017 Jul; 13(7):e1006913.
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Jnk1 Deficiency in Hematopoietic Cells Suppresses Macrophage Apoptosis and Increases Atherosclerosis in Low-Density Lipoprotein Receptor Null Mice. Arterioscler Thromb Vasc Biol. 2016 06; 36(6):1122-31.
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DRP1 inhibition rescues retinal ganglion cells and their axons by preserving mitochondrial integrity in a mouse model of glaucoma. Cell Death Dis. 2015 Aug 06; 6:e1839.
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The second window of desflurane-induced preconditioning is mediated by STAT3: role of Pim-1 kinase. Acta Anaesthesiol Scand. 2016 Jan; 60(1):103-16.
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Histone deacetylase 6 (HDAC6) promotes the pro-survival activity of 14-3-3ζ via deacetylation of lysines within the 14-3-3ζ binding pocket. J Biol Chem. 2015 May 15; 290(20):12487-96.
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BRAF inhibitor resistance mediated by the AKT pathway in an oncogenic BRAF mouse melanoma model. Proc Natl Acad Sci U S A. 2015 Feb 10; 112(6):E536-45.
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Coenzyme Q10 ameliorates oxidative stress and prevents mitochondrial alteration in ischemic retinal injury. Apoptosis. 2014 Apr; 19(4):603-14.
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Inhibition of cyclophilin D by cyclosporin A promotes retinal ganglion cell survival by preventing mitochondrial alteration in ischemic injury. Cell Death Dis. 2014 Mar 06; 5:e1105.