Pages that link to "Q33954324"
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The following pages link to Improved repair of dermal wounds in mice lacking microRNA-155 (Q33954324):
Displaying 19 items.
- The Role of MicroRNAs in Diabetic Complications-Special Emphasis on Wound Healing (Q26827627) (← links)
- Not miR-ly micromanagers: the functions and regulatory networks of microRNAs in mammalian skin (Q26829335) (← links)
- Transition from inflammation to proliferation: a critical step during wound healing (Q28068273) (← links)
- Exploring a Role for Regulatory miRNAs In Wound Healing during Ageing:Involvement of miR-200c in wound repair (Q33789656) (← links)
- In Vivo Inhibition of miR-155 Promotes Recovery after Experimental Mouse Stroke (Q36038242) (← links)
- Regulation of impaired angiogenesis in diabetic dermal wound healing by microRNA-26a (Q36609739) (← links)
- Non-Coding RNAs: New Players in Skin Wound Healing (Q37695147) (← links)
- An updated review of mechanotransduction in skin disorders: transcriptional regulators, ion channels, and microRNAs (Q38355578) (← links)
- Mutual interaction between BCL6 and miRNAs contributing to the pathogenesis of various cancers (Q38537155) (← links)
- New insights into microRNAs in skin wound healing (Q38644180) (← links)
- MicroRNA-155 Inhibition Promoted Wound Healing in Diabetic Rats (Q38691160) (← links)
- MicroRNA-155: a Novel Armamentarium Against Inflammatory Diseases (Q39040841) (← links)
- Interplay between miRNAs and human diseases (Q39127898) (← links)
- Emerging Roles for MicroRNAs in Diabetic Microvascular Disease: Novel Targets for Therapy (Q39190427) (← links)
- Emerging Roles for MicroRNAs in Diabetic Microvascular Disease: Novel Targets for Therapy (Q39315669) (← links)
- Silencing a Multifunctional microRNA Is Beneficial for Stroke Recovery. (Q52661746) (← links)
- microRNA-155 inhibition restores Fibroblast Growth Factor 7 expression in diabetic skin and decreases wound inflammation (Q64065244) (← links)
- The MicroRNA miR-155 Is Essential in Fibrosis (Q64104639) (← links)
- MicroRNA-34a deficiency leads to impaired wound closure by augmented inflammation in mice (Q94571775) (← links)