Pages that link to "Q28854318"
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The following pages link to Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway (Q28854318):
Displaying 50 items.
- Amyloid beta precursor protein (Q423510) (← links)
- Molecular topology as novel strategy for discovery of drugs with aβ lowering and anti-aggregation dual activities for Alzheimer's disease (Q21132276) (← links)
- Docosahexaenoic acid-derived neuroprotectin D1 induces neuronal survival via secretase- and PPARγ-mediated mechanisms in Alzheimer's disease models (Q21135583) (← links)
- Treatment of Alzheimer's disease with anti-homocysteic acid antibody in 3xTg-AD male mice (Q21142681) (← links)
- Transthyretin and the brain re-visited: is neuronal synthesis of transthyretin protective in Alzheimer's disease? (Q21202832) (← links)
- Cellular prion protein mediates impairment of synaptic plasticity by amyloid-beta oligomers (Q22251089) (← links)
- Gene transfer of human Apoe isoforms results in differential modulation of amyloid deposition and neurotoxicity in mouse brain (Q24308258) (← links)
- Characterizing affinity epitopes between prion protein and β-amyloid using an epitope mapping immunoassay (Q24313479) (← links)
- Homer2 and Homer3 interact with amyloid precursor protein and inhibit Abeta production (Q24316687) (← links)
- Progressive accumulation of amyloid-beta oligomers in Alzheimer's disease and in amyloid precursor protein transgenic mice is accompanied by selective alterations in synaptic scaffold proteins (Q24329069) (← links)
- Amyloid-β oligomers induce synaptic damage via Tau-dependent microtubule severing by TTLL6 and spastin (Q24337233) (← links)
- Inhibition of calpain prevents NMDA-induced cell death and beta-amyloid-induced synaptic dysfunction in hippocampal slice cultures (Q24603101) (← links)
- Apolipoprotein E receptors in the nervous system (Q24607165) (← links)
- S-Nitrosylation activates Cdk5 and contributes to synaptic spine loss induced by beta-amyloid peptide (Q24612303) (← links)
- Amyloid-beta-induced neuronal dysfunction in Alzheimer's disease: from synapses toward neural networks (Q24622558) (← links)
- Synthetic amyloid-beta oligomers impair long-term memory independently of cellular prion protein (Q24630045) (← links)
- Protein phosphatases and Alzheimer's disease (Q24632495) (← links)
- Mechanisms of synapse and dendrite maintenance and their disruption in psychiatric and neurodegenerative disorders (Q24632757) (← links)
- Relationships between expression of apolipoprotein E and beta-amyloid precursor protein are altered in proximity to Alzheimer beta-amyloid plaques: potential explanations from cell culture studies (Q24633831) (← links)
- Days to criterion as an indicator of toxicity associated with human Alzheimer amyloid-beta oligomers (Q24633883) (← links)
- Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory (Q24645499) (← links)
- Soluble oligomers of the amyloid beta-protein impair synaptic plasticity and behavior (Q24645994) (← links)
- Protein aggregation in the brain: the molecular basis for Alzheimer's and Parkinson's diseases (Q24650014) (← links)
- Reelin signaling antagonizes beta-amyloid at the synapse (Q24651028) (← links)
- Reversal of long-term dendritic spine alterations in Alzheimer disease models (Q24654995) (← links)
- Signaling effect of amyloid-beta(42) on the processing of AbetaPP (Q24656147) (← links)
- Synapses and Alzheimer's disease (Q26269868) (← links)
- Amyloidosis in Retinal Neurodegenerative Diseases (Q26739839) (← links)
- The Emergence of NMDA Receptor Metabotropic Function: Insights from Imaging (Q26741815) (← links)
- The Rationale for Insulin Therapy in Alzheimer's Disease (Q26744834) (← links)
- Non-ionotropic signaling by the NMDA receptor: controversy and opportunity (Q26745925) (← links)
- Calcium-Sensing Receptors of Human Neural Cells Play Crucial Roles in Alzheimer's Disease (Q26747527) (← links)
- Amyloid-β-Induced Dysregulation of AMPA Receptor Trafficking (Q26748723) (← links)
- Regulation of the Postsynaptic Compartment of Excitatory Synapses by the Actin Cytoskeleton in Health and Its Disruption in Disease (Q26748973) (← links)
- Emerging Link between Alzheimer's Disease and Homeostatic Synaptic Plasticity (Q26750576) (← links)
- Diabetes Mellitus Induces Alzheimer's Disease Pathology: Histopathological Evidence from Animal Models (Q26750648) (← links)
- Metals and Neuronal Metal Binding Proteins Implicated in Alzheimer's Disease (Q26765782) (← links)
- Beta-Amyloid and Tau-Protein: Structure, Interaction, and Prion-Like Properties (Q26765999) (← links)
- Dysfunction of NMDA receptors in Alzheimer's disease (Q26766185) (← links)
- Amyloid-β Receptors: The Good, the Bad, and the Prion Protein (Q26771532) (← links)
- Tau Oligomers: The Toxic Player at Synapses in Alzheimer's Disease (Q26773154) (← links)
- Genetic reduction of striatal-enriched tyrosine phosphatase (STEP) reverses cognitive and cellular deficits in an Alzheimer's disease mouse model (Q26781974) (← links)
- Gene expression parallels synaptic excitability and plasticity changes in Alzheimer's disease (Q26785375) (← links)
- Targeting the proper amyloid-beta neuronal toxins: a path forward for Alzheimer's disease immunotherapeutics (Q26822002) (← links)
- Wnts in action: from synapse formation to synaptic maintenance (Q26823548) (← links)
- The relevance of beta-amyloid on markers of Alzheimer's disease in clinically normal individuals and factors that influence these associations (Q26830467) (← links)
- Apolipoprotein E and apolipoprotein E receptors: normal biology and roles in Alzheimer disease (Q26858870) (← links)
- New pharmacological strategies for treatment of Alzheimer's disease: focus on disease modifying drugs (Q26866006) (← links)
- Inconsistencies and controversies surrounding the amyloid hypothesis of Alzheimer's disease (Q27003841) (← links)
- Molecular mechanisms of dendrite stability (Q27015653) (← links)