Endothelial cell signalling induced by trans-sialidase from Trypanosoma cruzi. (Q40098732)

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3 January 2020

title

Endothelial cell signalling induced by trans-sialidase from Trypanosoma cruzi (English)

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3 January 2020

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3 January 2020

Jose Osvaldo Previato

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3 January 2020

Lúcia Mendonça Previato

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3 January 2020

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Fernanda D Fajardo

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3 January 2020

Aurelio V Graça-Souza

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3 January 2020

Leonardo Freire-de-Lima

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3 January 2020

Fabiana Vieira

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3 January 2020

Murielle F Girard

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3 January 2020

Bernard Bouteille

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3 January 2020

Adriane R Todeschini

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3 January 2020

language of work or name

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2 August 2007

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3 January 2020

published in

Cellular Microbiology

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3 January 2020

volume

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issue

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3 January 2020

page(s)

88-99

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on focus list of Wikimedia project

3 January 2020

ScienceSource

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cites work

The crystal structure and mode of action of trans-sialidase, a key enzyme in Trypanosoma cruzi pathogenesis

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Thymocyte depletion in Trypanosoma cruzi infection is mediated by trans-sialidase-induced apoptosis on nurse cells complex

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The Trypanosoma cruzi trans-sialidase, through its COOH-terminal tandem repeat, upregulates interleukin 6 secretion in normal human intestinal microvascular endothelial cells and peripheral blood mononuclear cells

21 January 2018

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Nuclear factor (NF)-kappaB-regulated X-chromosome-linked iap gene expression protects endothelial cells from tumor necrosis factor alpha-induced apoptosis.

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Lysosome recruitment and fusion are early events required for trypanosome invasion of mammalian cells

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Costimulation of host T lymphocytes by a trypanosomal trans-sialidase: involvement of CD43 signaling

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Enzymatically inactive trans-sialidase from Trypanosoma cruzi binds sialyl and beta-galactopyranosyl residues in a sequential ordered mechanism.

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Trypanosome trans-sialidase targets TrkA tyrosine kinase receptor and induces receptor internalization and activation.

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