Heparan sulfate proteoglycan binding promotes APRIL-induced tumor cell proliferation. (Q34413225)

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11 January 2020

title

Heparan sulfate proteoglycan binding promotes APRIL-induced tumor cell proliferation (English)

1 reference

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heparan sulfate proteoglycans

11 January 2020

main subject

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1 reference

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11 January 2020

Hendriks J

1 reference

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11 January 2020

Planelles L

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11 January 2020

de Jong-Odding J

1 reference

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11 January 2020

Hardenberg G

1 reference

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11 January 2020

Pals ST

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11 January 2020

Hahne M

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11 January 2020

Medema JP

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11 January 2020

publication date

1 June 2005

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Cell Death & Differentiation

11 January 2020

published in

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11 January 2020

volume

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11 January 2020

issue

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11 January 2020

page(s)

637-648

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Biologically active APRIL is secreted following intracellular processing in the Golgi apparatus by furin convertase

11 January 2020

cites work

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The TNF family members BAFF and APRIL: the growing complexity

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BAFF AND APRIL: a tutorial on B cell survival

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The uncertain glory of APRIL.

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APRIL modulates B and T cell immunity

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DCs induce CD40-independent immunoglobulin class switching through BLyS and APRIL

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Impaired IgA class switching in APRIL-deficient mice

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APRIL, a new ligand of the tumor necrosis factor family, stimulates tumor cell growth

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7 January 2021

A soluble form of B cell maturation antigen, a receptor for the tumor necrosis factor family member APRIL, inhibits tumor cell growth

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7 January 2021

APRIL promotes B-1 cell-associated neoplasm.

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BAFF binds to the tumor necrosis factor receptor-like molecule B cell maturation antigen and is important for maintaining the peripheral B cell population

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APRIL and TALL-I and receptors BCMA and TACI: system for regulating humoral immunity

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Tumor necrosis factor (TNF) receptor superfamily member TACI is a high affinity receptor for TNF family members APRIL and BLyS

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7 January 2021

Interaction of the TNF homologues BLyS and APRIL with the TNF receptor homologues BCMA and TACI

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The crystal structure of a proliferation-inducing ligand, APRIL.

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Roles of heparan-sulphate glycosaminoglycans in cancer

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The hepatocyte growth factor/Met pathway controls proliferation and apoptosis in multiple myeloma.

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Heparan Sulfate-modified CD44 Promotes Hepatocyte Growth Factor/Scatter Factor-induced Signal Transduction through the Receptor Tyrosine Kinase c-Met

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7 January 2021

Heparin modulates the receptor-binding and mitogenic activity of hepatocyte growth factor on hepatocytes

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7 January 2021

Developmental changes in heparan sulfate expression: in situ detection with mAbs

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Heparan sulfate: growth control with a restricted sequence menu

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The cell-surface proteoglycan Dally regulates Wingless signalling in Drosophila

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7 January 2021

Specificities of heparan sulphate proteoglycans in developmental processes

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7 January 2021

The putative tumour suppressor EXT1 alters the expression of cell-surface heparan sulfate

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7 January 2021

Syndecan-1 is required for Wnt-1-induced mammary tumorigenesis in mice

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7 January 2021

Expression of c-Met and heparan-sulfate proteoglycan forms of CD44 in colorectal cancer

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Cell surface proteoglycan syndecan-1 mediates hepatocyte growth factor binding and promotes Met signaling in multiple myeloma

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Fibroblast Growth Factor-2 Stimulation of p42/44MAPKPhosphorylation and IκB Degradation Is Regulated by Heparan Sulfate/Heparin in Rat Mammary Fibroblasts

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