Targeted disruption of the Stat1 gene in mice reveals unexpected physiologic specificity in the JAK-STAT signaling pathway (Q29615968)

PubMed

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30 April 2017

Meraz MA

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30 April 2017

White JM

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30 April 2017

Sheehan KC

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30 April 2017

Bach EA

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30 April 2017

Rodig SJ

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30 April 2017

Dighe AS

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Kaplan DH

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Riley JK

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Greenlund AC

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30 April 2017

Campbell D

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30 April 2017

Carver-Moore K

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DuBois RN

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30 April 2017

Clark R

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30 April 2017

Aguet M

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30 April 2017

Schreiber RD

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language of work or name

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publication date

9 February 1996

1 reference

PubMed

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30 April 2017

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30 April 2017

1 reference

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30 April 2017

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30 April 2017

page(s)

431-42

1 reference

PubMed

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Requirement of endogenous interferon-gamma production for resolution of Listeria monocytogenes infection

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Multiple defects of immune cell function in mice with disrupted interferon-gamma genes

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Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins

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The proteins of ISGF-3, the interferon alpha-induced transcriptional activator, define a gene family involved in signal transduction

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Stat recruitment by tyrosine-phosphorylated cytokine receptors: an ordered reversible affinity-driven process

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Distinct regions of c-Mpl cytoplasmic domain are coupled to the JAK-STAT signal transduction pathway and Shc phosphorylation

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Contribution of STAT SH2 groups to specific interferon signaling by the Jak-STAT pathway

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An interleukin-4-induced transcription factor: IL-4 Stat

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Identification and purification of human Stat proteins activated in response to interleukin-2

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Immune response in mice that lack the interferon-gamma receptor

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Signaling by the cytokine receptor superfamily: JAKs and STATs.

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Interleukin 12 signaling in T helper type 1 (Th1) cells involves tyrosine phosphorylation of signal transducer and activator of transcription (Stat)3 and Stat4

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The control of interferon-inducible gene expression

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Interleukin-10-deficient mice develop chronic enterocolitis

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Tyrosine phosphorylation of DNA binding proteins by multiple cytokines

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Role of STAT2 in the alpha interferon signaling pathway

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High-frequency mutagenesis of human cells and characterization of a mutant unresponsive to both alpha and gamma interferons

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Epithelial immaturity and multiorgan failure in mice lacking epidermal growth factor receptor

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The protein tyrosine kinase JAK1 complements defects in interferon-alpha/beta and -gamma signal transduction

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Functional role of type I and type II interferons in antiviral defense

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Isolation of Large T Antigen-Producing Mouse Cell Lines Capable of Supporting Replication of Polyomavirus-Plasmid Recombinants

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Nitric oxide synthases: Roles, tolls, and controls

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Transcriptional responses to polypeptide ligands: the JAK-STAT pathway

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Proteins of transcription factor ISGF-3: one gene encodes the 91-and 84-kDa ISGF-3 proteins that are activated by interferon alpha

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Components of a Stat recognition code: evidence for two layers of molecular selectivity

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Activation of transcription by IFN-gamma: tyrosine phosphorylation of a 91-kD DNA binding protein

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Choice of STATs and other substrates specified by modular tyrosine-based motifs in cytokine receptors

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Complementation cloning of an MHC class II transactivator mutated in hereditary MHC class II deficiency (or bare lymphocyte syndrome)

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A protein tyrosine kinase in the interferon alpha/beta signaling pathway

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Complementation by the protein tyrosine kinase JAK2 of a mutant cell line defective in the interferon-gamma signal transduction pathway

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Maximal activation of transcription by Stat1 and Stat3 requires both tyrosine and serine phosphorylation

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The genomic structure of the STAT genes: multiple exons in coincident sites in Stat1 and Stat2

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Targeted disruption of gp130, a common signal transducer for the interleukin 6 family of cytokines, leads to myocardial and hematological disorders

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Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=8608597

PubMed publication ID

2 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017