Transformation of axial skeleton due to overexpression of bmi-1 in transgenic mice (Q24313654)

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English	Transformation of axial skeleton due to overexpression of bmi-1 in transgenic mice	scientific article

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scholarly article

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Transformation of axial skeleton due to overexpression of bmi-1 in transgenic mice (English)

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COMMD3-BMI1 readthrough

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GOA release 2020-03-11

segment specification

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GOA release 2020-03-11

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author name string

M J Alkema

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N M van der Lugt

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R C Bobeldijk

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A Berns

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M van Lohuizen

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language of work or name

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publication date

20 April 1995

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374

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6524

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724-7

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Novel zinc finger gene implicated as myc collaborator by retrovirally accelerated lymphomagenesis in E mu-myc transgenic mice.

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Drosophila genes Posterior Sex Combs and Suppressor two of zeste encode proteins with homology to the murine bmi-1 oncogene

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Sequence similarity between the mammalian bmi-1 proto-oncogene and the Drosophila regulatory genes Psc and Su(z)2.

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Imprinting a determined state into the chromatin of Drosophila

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Posterior transformation, neurological abnormalities, and severe hematopoietic defects in mice with a targeted deletion of the bmi-1 proto-oncogene

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Predisposition to lymphomagenesis in pim-1 transgenic mice: cooperation with c-myc and N-myc in murine leukemia virus-induced tumors

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Hoxb-4 (Hox-2.6) mutant mice show homeotic transformation of a cervical vertebra and defects in the closure of the sternal rudiments

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Homeosis in the mouse induced by a null mutation in the Hox-3.1 gene

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Homeotic transformations of murine vertebrae and concomitant alteration of Hox codes induced by retinoic acid

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Hox genes in vertebrate development

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Phenotypic consequences and genetic interactions of a null mutation in theDrosophila posterior Sex Combs gene

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Function of retinoic acid receptor gamma in the mouse

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Position effect variegation and chromatin proteins

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Related chromosome binding sites for zeste, suppressors of zeste and Polycomb group proteins in Drosophila and their dependence on Enhancer of zeste function

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