Occupational Health
Occupational Health
Syllabus
ofoccupational health and Ergonomics. Occupational Hazards and Occupational diseases
Role of
Definition
control of Occupational diseases. ESI Act ,1948 and The factories Act. 1948.
Examination
Prevention and bealth problems. The Merchant Shipping / Medical
Ayurveda in various Occupational
Rules 2000.
Learning objectives
The main object of ergonomics is to achieve environment which may be adverse to health are
the best mutual adjustment between the man and heat, cold, humidity, air, movement, heat radiation,
the machine, which are complimentary to cach light, vibrations, ionizing radiation etc. factors act
other, so that therc is increased efficiency, increascd in different ways singly and in different
oroduction and decreased accidents in the industry. combination
This term was coincd in a conference at
Chemical: These include large number of
Stockholm in 1961, conducted by International chemicals,
Ergonomics Association. Ergonomics has made a hazardous to toxic dusts and gases which are
the health of workers.
significant contribution in reducing the industrial
Bccidents and in overall health and efficiency of vial.Biological: Workers may come in contact to
the workers. rickettisal, bacterial and parasitic agents due
The health of the worker is influenced by 3 to close contact with animals or their products,
factors. Namely occupational (working) contaminated water, soil and food.
environment, domestic environment and social Man and Machine- The interaction between
security and welfare measures. man and machine. In almost all the industries, the
Burden of Occupational Diseases- There are machines are driven by power. Poor installation of
100 million occupational injuries causing 0.1 machines, the unguarded machines, protruding and
moving parts, poor maintenance etc. result in
million deaths in the world according to WHO. accidents. Working for long hours result in fatigue,
In India, it is estimated that 17 million (17% of discomfort and diseased efficiency.
global burden) occupational nonfatal injuries and Man and Man -The interaction between the
45,000 fatal injuries occur cach year. Out of 11
million cases of occupational diseases in the world.worker and his co-workers and the employer. This
1.9 million cases (17%) are contributed by India depends upon many psychosocial factors at work
and out of 0.7 million deaths in the world 0.12 place like nature of the work, colleagues,
million (17%) is contributed by India. communications, higher authority, system of
Aims :
payments, welfare conditions, work stability.
degree of responsibility, service condition, job
1. To increase the efficiencyi satisfaction, incentives etc.
2. To increase the production
3. To decrease the accidents Occupational Hazards
Objectives: An industrial worker is exposed to the following
1. To promote the health of the workers. five types of hazards (Physical, chemical,
2. To maintain the highest degree of physical, biological, mechanical and psychosocial hazards).
mental and social wel being of the workers. depending upon the nature of the occupation.
3. To prevent the discases by elimination of Physical Hazards
factors which are inimical to their health.
Occupational Environment Conditions- All . Heat: The effects are heat syncope, heat
the external conditions and influences, which is cramps, heat exhaustion, heat stroke, heat
hyperpyrexia, prickly heat, burns etc.
present at the work place and which influence the Cold: Frost bite, Reynaud's disease,
health of working personnel is occupational
environment. Man interacts with physical, chemical t erythromelalgia, erythrocyanosis, chilblains,
trench-foot, gangrene ctc.
and biological agent, machine and other men. . Heat syncope- It is fainting attack due to
Man and Physical, Chemical, and Biological pooling of blood in lower limbs.
agents
Physical: The physical factors in the working| Heat cranmps- It is painful and spasmodic
Swasthavritta evam Yoga
S52
joints of fingers, hands, clbows and shoulder
contractions of muscles due to loss of scodium may occur.
radiatlonExposur
and chloride.
Exposure to Utra-violet the skin- Darkenine
.Heat exhaustion- lt nmeans loss of salt leading to Ultra-iolet radiation-On
on to circulatory failure. thickening ofthe skin, erythema cancer
perpyrevia- t is characterized by ofthe skin,
skin. On the cyes (E.g. -Weldine)
.Heat hyT mechanisn1 without of the conjunctivitis,keratitis,,corneal ulcer,
failure in heat regulating Photophobin,
abot blindness (Welder's fish), snow blindness.
the features of heat stroke. Tenyerature is radiationExposure to
106 åF. lt may pceed tobeheat stroke. Exposure to lonizing radio-active isotopes
failure in the heat
" Heat stroke- There will
resulting in high onizing radiation- X-rays, 32 produce genetie
regulating mechanism,deliriun, convulsions, 60. phosphorus cancer, leukemia,
like cobaltmalformations,
temperature of the hody.consciousness. changes, extreme cases.
partial or total loss of pain are ulceration, sterility and death in
"Frost bite- Erythema and slight paraesthetic Chemical Hazards
features. In severe case painless,destruction, Local,
parts, result in decp seated Chemical agents act in 3 ways:depend on
gangrene and blistering may occur. Inhalation and Ingestion. The ill effects
Erythrocyanosis- Limbs are swollen and red duration of exposure, amount and individual
. susceptibility.
due to direct exposure to cold.
burning
.Chilblains- Erythema, itching.hecls, 1. Local effects: Dermatitis, eczema, ulcer and
especially of dorsa of fingers, toes,nose, even cancer.
Lesions can
cars on exposure to extreme cold. a. Denatitis due to allergy- machine oils, rubber,
be single or multiple and can become blistered X-ray, Caustic alkalies, lime etc.
and ulcerated.
b. Aromatic nitro and amino-compounds such
" Light- Effects occur either due to inadequate as TNT and aniline are absorbed through skin
light or excessive light. and cause systemic effects.
. Inadequate light- Headache, eye strain, eye 2. Inhalation: Dusts, fumes, gases and metals
fatigue, eye pain. and their compound.
.Excessive light- Bright light results in glaring. . Dusts-Dusts are fincly divided solid particles
visual fatigue, blurring of vision, discomfort with size ranging from 0.1 to 150 microns.
and accidents.
. Noise- Noise is a health hazard in many They are released into the atmosphere during
industries. The effects of noise are of two types. erushing, grinding, abrading, loading and
NoseAuditory effects Deafness (emporary unloading operations. Dusts are produced in a
or permanent), tinnitus (buzzing in the cars). number of industries nmines, foundry, quary.
Non-Auditory effects- Fatigue, irritability. pottery, textile, wood or stone working
nervousness, interference with speech and industries, Example- Paeumoconiosis.
communication, hypertension, peptic ulcer, Fumes- Metal fume fever (This chemical
higher environmental stress. intoxication results from inhalation of fumes
. VibrationVibration- Frequency range 10 of molten metals like arsenic, antimony.
500Hz, when working with tools like drill, beryllium, cadmium, cobalt, lead, zinc,
hammers etc. vibration generally affects hands mercury tc.)
and arms. Afer months and yeas of "Gases- Exposure to gases is acommon hazard
fine blood vessels of fingers mayexposure,
become in industries.
increasingly sensitive to spasm, injuries to a. Simple gases- O2, H2
Occupationnl Health 553
h. Asphyxiating gases- Co,
methyl isocyanides gas ctc. s02, C12, H2S, e) Noise-Occupational deafness.
e. Anaesthetic gases- Chloroform, ether, 9Radintion- Cancer, leukemia, aplasticanemia,
trichloroethylene pancytopenin.
Ingestion: Toxie hazards occurring from the g) Mechanical factors- Injuries, accidents.
octals like lead, arsenic, h) Electricity- Burns.
manganesc, chromium ete, mercury, cadmium, 2. Diseases due to chemical agents
Biological Hazards a) Gases- CO2, CO, HCN,CS2, NH3, N2, H2S,
HCI, SO2 these cause gas poisoning.
Workers may be exposed to infective and b) Dusts (Pneumoconiosis)
parasitic agents at the place of work. These are from L Inorganie dusts
the animals and soil. These are common in
agricultural industry,. " Coal dust- Anthracosis
From the animals- they are called 'Zoonotic " Silica- Silicosis
diseases'. Ex: Anthrax, rabies, plague, "Asbestos- Asbestosis, cancer lung
salmonellosis, bovine tuberculosis. . Iron- Siderosis
. From the soil- Tetanus, I. Organic (Vegetable) dusts
gag-gangrene,
malignant oedema, anthrax, mycetoma. " Cane fiber- Bagassosis
Mechanical Hazards- 10% of accidents in the " Cotton dust- Byssinosis
industry are due to mechanical causes. " Tobacco- Tobacossis
Psyehological Hazards- These are due to .Hay or grain dust- Farmer's lung
failure of the worker to develop a healthy II. Metals and their compounds-Toxic hazards
relationship with his co-workers, employers, from lead, mercury, cadmium, manganese,
management, supervisors etc. They are divided into berylium, arsenic, chromium etc.
two groups. IV. Chemicals- Acids, alkalis, pesticides
1. Psychological and behavioural changes: Such V. Solvents- Carbon bisulphide, benzene,
as hostility, aggressiveness, anxiety, trichloroethylene, chloroform etc.
depression, frustration, tardiness, alcoholism, 3. Diseases due to biological agents
drug addiction, sickness absenteeism etc. Brucellosis, leptospirosis, anthrax, actino
2. Psychosomatic ill health: Such as neurosis, mycosis, hydatidosis, psittacosis, tetanus,
fatigue, propensity to peptic ulcer, encephalitis, fungal infcctions ctc.
hypertension, asthma etc. 4. Occupational cancers- Cancer of skin, lungs,
bladder.
Occupational Diseases and their Preve
ntion and Control
5. Oceupational dermatitis- Dematitis, eczema.
6. Diseases of psychological origin- Industrial
1. Diseases due to physical agents neurosis, hypertension, peptic ulcer ete.
a) Heat- Heat hyperpyrexia, heat exhaustion, heat Occupational Dermatitis
syncope, heat cramps, burns and local effects
such as prickly heat. Occupational dermatitis is a big health problem
b) Cold- Trench foot, frost bite, chilblains. in many industries. The causes may be:
c) Light- Occupational cataract, miner's 1. Physical- Heat, cold, moisture, friction,
nystagmus. pressure, X-rays and other rays.
d) Pressure- Caisson disease, air embolism, blast 2. Chemical- acids, alkalies, dyes, solvents,
(explosion). grease, tar, pitch, chlorinated phenols.
554 Swasthavrita evan Yoga
Treatment : Silicosis is an itreversible Treatment: There is no cure for the black lung
= oce. Trestment options curently disease. Treatrments are aimed at the symptoms and
focs cm aleviting
apiaices Thesethe sypeos and preenting complicated.
inctade: Prevention and control:
L Saogping furtber exposure to siica and other 1. Dust control measures like wearing face mask
ng iritts, inclading tobacco. 2. Improving the ventilation of the factory
2 Coegh ppressnts
3 Atibicis for bacterial lung infection. 3. Improving the machinery to reduce the dust
4. Wetting procedures should be carried out so
4 TB prophylaxis for those with positive that the dust will not concentrated in air.
oberclosis skin test Measures of Health Protection in Industrial units
s Ches ptysiotherapy to betp the broachial
dainage of macus. 1. Proper Nutrition
6 Orygen administration to treat hypoxemia, if 2. Communicable disease control
rset Broochodilators to failitate breathing. 3. Enviroment sanitation
7. Lng Mental bcalth promotion
& Trnplattion 5. Health education
6. Protective measures foe women and children
Prevention and control:
7. Family planning
.Rigoroas dast control measures like wearing
mesk improving the vetilation of work place. Byssinosis
.Warspray is often nsed where dust emanates
Byssinosis is a discase of the lungs brought on
" Dast can also be controlled by dry air filtering by breathing in cotton, dust or dusts from other
Anthracosis (Coal Workers vegetable fibres such as flax, hemp or jute while at
Pneumonconiosis) work.
Incidence: In United States, more than 35000
Black lung diseasc, also known as Coal textile workers have been disabled by Byssinosis
worker's pneumoconiosis, is caused by long and 183 died between 1979 and 1992. In India,
exposure to coal dust. It is acondition characterized among 35% of textile industry 7to 8% of workers
by the accumulation of carbon in lungs. are affected with Byssinosis.
Causes: Inhalation of coal dust. Causes:
Risk Factors: Smoking . The most common cause is breathing in the
Signs & Symptoms: No carly symptoms, dust produced by raw cotton.
Cough. Chest pain, Breathing difficulty. Dyspnoca.
Bronchitis. Cyanosis, Progressive lung stiffening.
. People who work in the textile industry
. Smoking increases the risk for this disease.
Shortness of breath, Impaired lung functions Signs and Symptoms :
Diagnosis: . Chest tightness
1. History collection . Progressive Dyspnoea
2. Physical examination Chronic Cough
3. Chest X-ray . Tachypnea
4. Pulmonary function tests . Wheezing
5. Chest CT Scan
6. HRCT- High resolution CT Scan
" Symptoms will get worse at the beginning of
the work week and then improve while you
Swasthavritta evam Yoga
556
6. Wetting procedures so that the dust will not
are away from the work place, or late in the concentrated in the air.
work weck.
Diagnosis : Bagassosis
1. History collection: Occupation and will ask
many qucstions to try to find out whether
your Bagassosis is the name given to an occupational
relate to certain exposure or times disease of the lung caused by inhalation of baat
symptoms or sugarcane dust. It was first reported in India by
of exposure. Ganguli and Pal in 1955 in a cardboard
2. Physical examination manufacturing firm near Kolkata. India has a laree
3. Chest X-ray
function tests- Shows typicalcane-sugar industry.
4. Pulmonaryobstruction and a reduction Symptoms Breathlessness, cough.
airflow
investigatory capacity, especially if measured haemoptysis and slight fever. Initially there is acta
at the start and end of the first work shift. diffuse bronchiolitis.
Treatment : Diagnosis- Skiagram may show mottling in
1. The most important treatment is to remove the lungs or shadow. There is impairment of pulmonary
function.
source of exposure to the offending agent.
2 Medications such as bronchodilators will If treated early, there is resolution of the acute
usually improve symptoms. inflammatory condition of the lung. If left untreated,
3. Coricosteroids may be preseribed in more there is diffuse fibrosis, emphysema and
bronchiectasis.
severe cases.
4. Stopping smoking is very important for people Prevention and control
with this condition. 1. Dust control - Measures for the prevention
5. Respiratory treatments including Nebulizers and suppression of dust such as wet process,
and postural drainage for chronic Conditions. enclosed apparatus, exhaust ventilation etc.
6. Home oxygen therapy if low blood oxygen should be used.
levels are detected.
2. Personal Protection- Personal protective
7. Physical exercise programs, breathing
exercises and patient education programs are equipment (masks or respirators with
often very helpful for people with a chronic mechanical filters or with oxygen or air supply)
lung disease. may be necessary.
3. Medical Control- Initial medical examination
Complications : Chronic lung disease and and periodical medical check-ups of the
Emphysema.
Prevention and control: workers are indicated.
4. Bagasse Control- By keeping the moisture
1.Controlling dust to prevent the content above 20 per cent and spraying the
disease. occurrence of bagasse with 2per cent propionic acid, awidely
2. Using face masks
the airway. prevent the dust entering used fungicide, bagasse can be rendered safe
for manufacturing use.
3. Improving
the dust is ventilation of the factory so that
reduced. Asbestosis
4.Reduction of dust levels by improving
machinery. Asbestosis is a chronic inflammatory and
3. Stop smoking if you are a textile fibrotic medical condition affectingtne
worker. industryparenchymal tissue of the lungs caused by
inhalation and retention of asbestos fibres.
Occupational Health 557
Causes:
2. Dust control measures like wearing mask,
1. Exposure to asbestos fibers wetting procedures, ventilation should be
2. Occupation like mining, manufacturing, improved in the work place
handling or removal of asbestos are at risk of 3. Frequent rest periods
developing asbestosis.
4. Usage of sophisticated machines.
3. Smoking increases the risk of developing
asbestosis.
Farmer's Lung
Signs and symptoms:
Farmer's lung is due to the inhalation of mouldy
1.Dyspnoca especially on exertion hay or grain dust in the agricultural field. In grain
2. Chest pain dust or hay with a moisture content of over 30
3. Cough, crackles present percent, bacteria and fungi grow rapidly, causing
Possible additionalsymptoms include : a rise of temperature to 40 to 50 deg.C. This heat
1. Nail abnormalities encourages the growth of thermophilic
2. Clubbing of fingers actinomycetes, of which Micropolyspora faeni is
Diagnosis: the main cause of farmer's lung.
Symptoms- The acute illness is characterized
1. History collection about the occupation and
will ask many questions to try to find out by general and respiratory symptoms and physical
whether you relate to occupation signs. Repeated attacks cause pulmonary fibrosis
2. Physical examination
and inevitable pulmonary damage and cor
pulmonale. It is quite possible that this condition
3. Chest X - ray shows lung changes might be widespread in India considering the bulk
4.CT scan of the lungs shows the specific areasof the population engaged in agricultural work.
affected Diagnosis- X"ray chest shows 'fine nodular
5. Gallium lung scan shows the specific areas density
affected
6. Pulmonary function tests show typical airway Prevention and control
obstruction and a reduction in ventilator 1. Dust control measures like wearing mask,
cover the mouth and nose by cloth.
capacity.
Treatment:
2. Frequent rest periods.
1. There is no cure available List of Pnemoconiosis