TAGUM DOCTORS COLLEGE INC.
Mahogany St., Rabe Subdivision Tagum City
Bachelor of Science in Nursing
DRUG STUDY
Name of Student : Sian, Liza P. – BSN3, Group 1
Name of Client : Estrera, Maxima Age: 81 y.o. Sex: Female Rating:
Date of assessment: Nov. 08, 2018 Physician: Dr. Jampayas Status: Married
Chief complaint : Infected right foot Admitting diagnosis: Infected Right Foot t/c Sepsis
Date Image of Drug Name of Drug Classification Dosage/ Mechanism of Action
Time/Route
Nov. 08, Brand name: Therapeutic: 1 tab Pharmacodynamics:
2018 Ketosterile Supplements PO The plasma kinetics of amino acids and their integration in metabolic
TID pathways are well established. It should nevertheless be noted that
Generic name: Pregnancy in uremic patients, the plasma disturbances do not seem to depend
Ketoanalouge Category Risk: on digested amino acid intake and that the post absorptive kinetics
Not seems to be distributed very early in the development of the
established disease. In normal individuals, there is an increase in the plasma
level of ketoanalogues, 1 min after oral ingestion. These levels reach
values that are approximately 5 times higher than the initial level.
Peak levels are reached within 20-60 min and normal levels are
reached again after 90 min. Gastrointestinal absorption is thus very
rapid. In the plasma a simultaneous increase in levels of the
ketoanalogue and the corresponding amino acid show that
transamination of the ketoanalogues are very rapid. Due to natural
pathways of disposal of a-ketonic acids in the organism, it is
probable that exogenous intakes are very rapidly integrated into
metabolic cycles. Ketoacids follow the same catabolic pathways as
the classical amino acids. No specific study on ketoacid excretion
has been performed to date.
Pharmacokinetics:
Absorption: absorption in the gastrointestinal tract!
Distribution: widely distributed
Metabolism Excretion: metabolized by the liver, excreted by the
liver
Indication Contraindications Adverse Reaction Nursing Responsibilities
General Indication: Hypercalcemia, disturbed Hypercalcemia may Before:
Prevention and therapy of amino acid metabolism. In develop. 1. Assess electrolyte levels
damages due to faulty or case of hereditary 2. Explain therapeutic value of drug
deficient protein metabolism phenylketonuria, it has to be 3. Assess allergy to the drug
in chronic renal insufficiency taken into account that 4. Caution patient of the different side effects
in connection with limited ketoanalogue contains 5. Assess vital signs
protein food of less than or phenylalanine. Disturbed 6. Proper preparation of the drug
equal to 40g/day for adults. amino acid metabolism. During:
7. Verify patient’s identity
8. Administer with food to prevent GI upset
9. Administer drug at right time, route, and dosage
10. Advise to swallow the tablet whole
11. Monitor vital signs
After:
12. Document administration of drug
13. Instruct patient to report immediately if symptoms of
hypercalcemia occurs like muscle weakness,
constipation
14. Monitor calcium levels
15. Monitor for signs of hypercalcemia and electrolyte
level
16. Monitor vital signs especially cardiac changes.
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