Pages that link to "Q35080459"
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The following pages link to Activation of group II metabotropic glutamate receptors promotes DNA demethylation in the mouse brain (Q35080459):
Displaying 31 items.
- Genetic deletion of Gadd45b, a regulator of active DNA demethylation, enhances long-term memory and synaptic plasticity (Q24600904) (← links)
- Epigenetic RELN Dysfunction in Schizophrenia and Related Neuropsychiatric Disorders (Q26752594) (← links)
- Advancements in the Underlying Pathogenesis of Schizophrenia: Implications of DNA Methylation in Glial Cells (Q26773159) (← links)
- Current status and future prospects for epigenetic psychopharmacology (Q26825464) (← links)
- A Methionine-Induced Animal Model of Schizophrenia: Face and Predictive Validity (Q27300760) (← links)
- Metabotropic Glutamate 2/3 Receptors and Epigenetic Modifications in Psychotic Disorders: A Review (Q28070122) (← links)
- Treatment-resistant schizophrenia: current insights on the pharmacogenomics of antipsychotics (Q28079065) (← links)
- Growth arrest and DNA-damage-inducible, beta (GADD45b)-mediated DNA demethylation in major psychosis (Q28394929) (← links)
- Gadd45b is an epigenetic regulator of juvenile social behavior and alters local pro-inflammatory cytokine production in the rodent amygdala (Q30651420) (← links)
- Epigenetic mechanisms during ageing and neurogenesis as novel therapeutic avenues in human brain disorders. (Q33858861) (← links)
- Potential molecular and cellular mechanism of psychotropic drugs (Q34129880) (← links)
- Upregulation of TET1 and downregulation of APOBEC3A and APOBEC3C in the parietal cortex of psychotic patients (Q34297541) (← links)
- Pharmacological activation of group-II metabotropic glutamate receptors corrects a schizophrenia-like phenotype induced by prenatal stress in mice. (Q35760210) (← links)
- Gadd45b knockout mice exhibit selective deficits in hippocampus-dependent long-term memory. (Q36126222) (← links)
- The dynamics of DNA methylation in schizophrenia and related psychiatric disorders. (Q36465820) (← links)
- DNA Methylation of BDNF Gene in Schizophrenia. (Q36567277) (← links)
- Epigenetic control of neurotransmitter expression in olfactory bulb interneurons (Q36756118) (← links)
- Prenatal stress induces schizophrenia-like alterations of serotonin 2A and metabotropic glutamate 2 receptors in the adult offspring: role of maternal immune system (Q37013377) (← links)
- Epigenetics of psychoactive drugs (Q38040133) (← links)
- Epigenetic changes brought about by perinatal stressors: a brief review of the literature (Q38043830) (← links)
- Ing1 functions in DNA demethylation by directing Gadd45a to H3K4me3. (Q39198445) (← links)
- DMSO is a strong inducer of DNA hydroxymethylation in pre-osteoblastic MC3T3-E1 cells (Q39363935) (← links)
- DNA methylation/demethylation network expression in psychotic patients with a history of alcohol abuse. (Q45927211) (← links)
- Nicotine restores Wt-like levels of reelin and GAD67 gene expression in brain of heterozygous reeler mice (Q46314942) (← links)
- DNA methylation signatures of mood stabilizers and antipsychotics in bipolar disorder (Q47593705) (← links)
- Sex-Specific Effects of Prenatal Stress on Bdnf Expression in Response to an Acute Challenge in Rats: a Role for Gadd45β. (Q49137999) (← links)
- DNA Methylation as a Biomarker of Treatment Response Variability in Serious Mental Illnesses: A Systematic Review Focused on Bipolar Disorder, Schizophrenia, and Major Depressive Disorder (Q57027559) (← links)
- Epigenetic Alterations in Prenatal Stress Mice as an Endophenotype Model for Schizophrenia: Role of Metabotropic Glutamate 2/3 Receptors (Q60048499) (← links)
- Impaired DNA demethylation of C/EBP sites causes premature aging (Q89046872) (← links)
- Dissecting the Epigenetic Changes Induced by Non-Antipsychotic Mood Stabilizers on Schizophrenia and Affective Disorders: A Systematic Review (Q94544668) (← links)
- Metabolomic and transcriptomic signatures of prenatal excessive methionine support nature rather than nurture in schizophrenia pathogenesis (Q98177107) (← links)