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Cardiology & Ekgs: Archer Nclex Review

The document provides an overview of cardiology and EKGs for the NCLEX exam. It discusses anatomy and physiology concepts like cardiac output, preload, afterload, and contractility. Pathologies like coronary artery disease, myocardial infarction, heart failure, hypertension, and arrhythmias are explained. Treatment options for these conditions focus on decreasing workload on the heart through medications, lifestyle changes, and management of symptoms.

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daphne farley
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100% found this document useful (1 vote)
888 views59 pages

Cardiology & Ekgs: Archer Nclex Review

The document provides an overview of cardiology and EKGs for the NCLEX exam. It discusses anatomy and physiology concepts like cardiac output, preload, afterload, and contractility. Pathologies like coronary artery disease, myocardial infarction, heart failure, hypertension, and arrhythmias are explained. Treatment options for these conditions focus on decreasing workload on the heart through medications, lifestyle changes, and management of symptoms.

Uploaded by

daphne farley
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as PDF, TXT or read online on Scribd
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CARDIOLOGY & EKGS

ARCHER NCLEX REVIEW


MUST KNOW SYSTEM FOR NCLEX SUCCESS!
ARCHER SLIDES ARE TO BE USED WITH RAPID FIRE/ CRASH COURSE TO
UNDERSTAND FULL CONCEPTS & APPLY IN NCLEX QUESTION SCENARIOS.
RAPID PREP WILL COVER THESE SLIDES AND EXPLAIN HIGHYIELD
CONCEPTS IN DETAIL
2 HOURS LIVE WEBINAR ON NOVEMBER 3RD, 2020
REGISTER AT WWW.ARCHERREVIEW.COM
ANATOMY & PHYSIOLOGY
BLOOD FLOW THROUGH THE HEART
HEMODYNAMICS
● Preload
○ Amount of blood returning to right side of the heart
● Afterload
○ Pressure against which the left ventricle must pump to eject blood
● Compliance
○ How easily the heart muscle expands when filled with blood
● Contractility
○ Strength of contraction of the heart muscle
● Stroke volume
○ Volume of blood pumped out of the ventricles with each contraction
● Cardiac output
○ The amount of blood the heart pumps through the circulatory system in a minute
CARDIAC OUTPUT
WHY is cardiac output SO important?!

● Tissue perfusion!
● End organ function
● Delivery of oxygen and nutrients to each and every cell in the body!
● Poor cardiac output??
○ Decreased LOC (not enough blood flow to the BRAIN)
○ Chest pain, weak peripheral pulses (not enough blood flow to the HEART)
○ SOB, crackles, rales (not enough blood flow to the LUNGS)
○ Cool, clammy, mottled extremities (not enough blood flow to the SKIN)
○ Decreased UOP (not enough blood flow to the KIDNEYS)

CO = SV X HR
CAUSES OF CAUSES OF
DECREASED CO INCREASED CO
● Bradycardia
● Arrhythmias ● Increased blood volume...sometimes
○ Pulseless v-tach ● Tachycardia...sometimes
○ V-fib ● Medications
○ Asystole ○ ACE Inhibitors
○ SVT ○ ARBS
● Hypotension ○ Nitrates
● MI ● Inotropes
● Cardiac muscle disease
EKG

ARCHER REVIEW CRASH COURSE


P-wave: normal
PR Interval: 0.12-0.20
QRS: <0.12
Rate: 60-100
Regularity: Regular

Normal Sinus Rhythm


P-wave: Normal Causes:
PR Interval: 0.12-0.20 -Sleep
QRS: <0.12 -Inactivity
Rate: <60 -Very athletic
Regularity: Regular -Drugs
-MI

Sinus Bradycardia
P-wave: Normal Causes:
PR Interval: 0.12-0.20 -Caffeine
QRS: <0.12 -Exercise
Rate: >100 -Fever
Regularity: Regular -Anxiety
-Drugs
-Pain
Sinus Tachycardia -Hypotension
-Volume depletion
P-wave: “saw-tooth” Causes:
PR Interval: none -Heart disease
QRS: <0.12 -MI
Rate: 250-400 -CHF
Regularity: Regular or -Pericarditis
Irregular

Atrial Flutter
P-wave: ‘wavy’ Causes:
PR Interval: none -Heart Disease
QRS: <0.12 -Pulmonary Disease
Rate: >400 -Stress
Regularity: irregular -Alcohol
-Caffeine

Atrial Fibrillation
P-wave: hidden Causes:
PR Interval: -Caffeine
immeasurable -CHF
QRS: <0.12 -Fatigue
Rate: 150-250 -Hypoxia
Regularity: Regular -Altered pacemaker in heart

Supraventricular Tachycardia (SVT)


Causes:
P-wave: none -MI
PR Interval: none -Ischemia
QRS: >0.11 - ‘wide -Digoxin toxicity
complex & bizarre’ -Hypoxia
Rate: 150-250 -Acidosis
Regularity: Regular -Hypokalemia
-Hypotension
Ventricular Tachycardia (V-Tach)
Causes:
-MI
P-wave: none
-Ischemia
PR Interval: none
-Hypoxia
QRS: none
-Acidosis
Rate: none
-Hypokalemia
Regularity: Irregular
-Hypotension
-Most common cause of sudden
death
Ventricular Fibrillation (V-fib)
P-wave: possible to Causes:
have some random p- -Follows VT/VF in cardiac
waves arrest
PR Interval: none -Acidosis
QRS: none -Hypoxia
Rate: none -Hypokalemia
Regularity: n/a -Hypothermia

Asystole -Overdose
CORONARY ARTERY DISEASE
(CAD)
WHAT IS CORONARY ARTERY DISEASE?
● The most common type of cardiovascular disease.
● Includes two types
○ Chronic stable angina
○ Acute coronary syndrome (aka MI)
CHRONIC STABLE ANGINA
● Chronic disease caused by narrowing of coronary
arteries and plaque build up.
● There are periods of decreased blood flow to the
heart muscle
● Decreased blood flow leads to decreased oxygen,
and ischemia.
● Ischemia causes chest pain
TREATMENT
● Nitroglycerin
○ Venous and arterial dilation → decreased afterload → increased CO
○ Given sublingual
○ Administer 1 pill q5 minutes for 3 doses
○ Do not swallow
○ Keep in a dark bottle in dry, cool place
○ Expected side effect = headache
EDUCATION
● DECREASE THE WORKLOAD OF THE HEART!
○ Rest
○ Do not overeat
○ No caffeine
○ Avoid temperature extremes
○ No smoking
○ Promote weight loss
○ Reduce stress
MYOCARDIAL INFARCTION
(MI)
WHAT IS A MYOCARDIAL INFARCTION
Myocardial infarction = acute coronary syndrome = unstable angina

● There is decreased blood flow to the heart, leading to decreased oxygen, and not only ischemia, but also
necrosis.
● Goal is to act quickly and limit the damage.
ASSESSMENT
● Chest pain
○ Crushing
○ Radiating to left arm or jaw
○ Between shoulder blades
● Epigastric discomfort/indigestion
● Fatigue
● SOB
● Vomiting
LABS
● CPK-MB
○ Cardiac specific isoenzyme
○ Increases with damage to cardiac cells
● Troponin
○ Cardiac biomarker
○ Increases with myocardial damage
● Myoglobin
○ Protein in muscle cells
○ Increases with damage to ANY muscle cell
○ Negative results can help rule out an MI, but positive results are not specific.
TREATMENT
● Cath lab within 90 minutes for PCI
○ Especially important if it’s a STEMI!
● Oxygen
● Aspirin
● Nitroglycerin
● Morphine
EDUCATION
● Quit smoking
● Increase activity gradually
● Diet
○ Low fat
○ Low salt
○ Low cholesterol
● Exercise
○ Avoid isometric exercises
○ Walking is a good choice
HEART FAILURE
WHAT IS HEART FAILURE?
The inability of the heart muscle to pump enough blood to meet the body's needs for blood and oxygen.

● Often results as a complication of other diseases


● #1 cause of HF is hypertension
● Other causes:
○ Cardiomyopathy
○ Endocarditis
○ MI
● Two types: Left and Right
LEFT-SIDED HEART FAILURE
Left side of the heart cannot move blood forward to the body.

Blood is backing up in the LUNGS.

Assessment:

● Pulmonary congestion
● Wet lung sounds
● Dyspnea
● Cough
● Blood tinged sputum
● S3
● Orthopnea
RIGHT HEART FAILURE
Right side of the heart cannot move blood forward to the lungs.

Blood is backing up in the BODY.

Assessment:

● Jugular venous distention


● Dependent edema
● Hepatomegaly
● Splenomegaly
● Ascites
● Weight gain
● Fatigue
● Anorexia
TREATMENT
● DECREASE THE WORKLOAD OF THE HEART!
● Primary strategy is to decrease afterload:
○ ACE Inhibitors
■ Arterial dilation→ decreased afterload → Increased stroke volume
○ ARBS
■ Decrease BP → decreased afterload → Increased CO
● Increase contractility
○ Digoxin
● Diuresis
○ Pt needs help reducing excess fluid
EDUCATION
● Take diuretic medications in the AM
● Monitor electrolyte levels while on diuretics
● Low sodium diet
○ This helps decrease fluid
● Elevate the HOB
○ Will help with diuresis
● Daily weight
○ Same time
○ Same scale
○ Same clothes
● Report any increase of 2-3 lbs in one day
HYPERTENSION
WHAT IS HYPERTENSION?
High blood pressure!

Normal <120/80
Elevated 120-129/80
Hypertension >130/>80
Hypertensive Crisis >180/>120
CAUSES & RISK FACTORS
● Family history
● African american race
● Increased age
● Obesity
● HLD
● CAD
● Stress
● Smoking
● High salt intake
● Caffeine
ASSESSMENT
● Often asymptomatic until very severe
● Vision changes
● Headaches
● Dizziness
● Nosebleeds
● SOB
● Angina
COMPLICATIONS
● Stroke
● MI
● Renal Failure
● Heart Failure
● Vision loss
TREATMENT & EDUCATION
● Medications
○ ACE inhibitors
○ Beta Blockers
○ CCB
○ Diuretics
● Diet
○ DASH
○ Low salt
○ Avoid caffeine and alcohol
○ Weight loss
○ Smoking cessation
● Lifestyle
○ Less sitting more walking
SHOCK
WHAT IS SHOCK??
● A state where the vital organs are not receiving adequate oxygenation.
● This lack of oxygenation causes organ damage and forces the cells to use anaerobic metabolism to create
energy….producing lactate.

● Cardiovascular system is composed of:


○ The blood
○ The vasculature
○ The heart
● A disruption in any of these three components can cause a lack of oxygen delivery to the organs, causing shock.
● Which component is ‘broken’ determines the type of shock.
Hypovolemic
TYPES OF SHOCK Cardiogenic
Distributive
HYPOVOLEMIC SHOCK
PATHOPHYSIOLOGY
● Low blood flow
● There is a loss of the circulating volume
● Not enough blood to enter the heart (preload), which
decreases cardiac output.
● The body will vasoconstrict to compensate.
CAUSES
● Hemorrhage
● Traumatic injury
● Dehydration
○ Vomiting
○ Diarrhea
● Burns
ASSESSMENT
● Compensation
○ Weak
○ Pale
○ Tachycardic
○ Anxious
● Failing
○ Hypotension
○ Weak pulses
○ Tachycardic
○ Decreased LOC
○ Pale
○ Cool
○ Clammy
○ Decreased UOP
TREATMENT
● Fix the cause
○ Stop vomiting/diarrhea
○ Stop bleeding
■ Repair in OR
● Replace volume
○ Isotonic IVF
■ NS
■ LR
○ Blood products
● Support perfusion
○ Vasopressors
CARDIOGENIC SHOCK
PATHOPHYSIOLOGY
● The heart fails to pump sufficient blood out to the organs
● “Pump failure”
● Something is stopping the heart itself from getting blood out to the body
● Without sufficient blood delivered to the body, there is inadequate oxygenation
● Lack of oxygen impairs normal cellular metabolism
CAUSES
● MI
● Cardiac tamponade
● Pulmonary embolism
ASSESSMENT
● Decreased perfusion
○ Hypotension
○ Weak pulses
○ Cool, pale, clammy
○ Decreased UOP
○ Decreased LOC
● Volume overload
○ JVD
○ Crackles
○ SOB
○ Muffled heart sounds
○ S3
TREATMENT
● TREAT THE CAUSE ● IABP
○ MI ● LVAD
■ PCI ● Transplant
■ CABG
○ PE
■ Thrombolytics
○ Tamponade
■ Pericardiocentesis
● Improve contractility
○ Dopamine
○ Dobutamine
● Decrease afterload
○ Diuretics
○ Dobutamine
DISTRIBUTIVE SHOCK
PATHOPHYSIOLOGY
● Something causes an immune or autonomic response in the body
● This alters vascular tone
● The result is massive peripheral vasodilation
● With so much vasodilation, the blood pressure is inadequate to provide blood flow to the vital organs.
● Without sufficient blood delivered to the body, there is inadequate oxygenation
● Lack of oxygen impairs normal cellular metabolism
CAUSES
● Anaphylactic
○ Allergic reaction
● Neurogenic
○ SCI
● Septic
○ Systemic infection
○ Causes release of inflammatory cytokines
ASSESSMENT
Specific:
● Decreased oxygen
● Hypotension ● Anaphylactic
● Tachycardia ○ Hives
● Tachypnea ○ Rash
● Warm, flushed skin ○ Swelling
● Decreased LOC ○ Wheezing
● Neurogenic
○ SCI
○ Priapism
● Septic
○ Hyperthermic
○ Infection
TREATMENT
● Anaphylactic
○ Epinephrine
○ Corticosteroids
○ Bronchodilators
● Neurogenic
○ Cooling
○ Supportive care
● Septic
○ IV antibiotics
○ IVF

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