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Pediatric Nursing Reviewer Cardiovascular

This document summarizes several types of pediatric cardiovascular dysfunctions including fetal circulation, congenital heart defects like atrial septal defect (ASD) and ventricular septal defect (VSD), obstructive defects like coarctation of the aorta, and acquired heart diseases such as rheumatic heart disease and bacterial endocarditis. It describes the pathophysiology, manifestations, and interventions for each condition.

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0% found this document useful (1 vote)
1K views4 pages

Pediatric Nursing Reviewer Cardiovascular

This document summarizes several types of pediatric cardiovascular dysfunctions including fetal circulation, congenital heart defects like atrial septal defect (ASD) and ventricular septal defect (VSD), obstructive defects like coarctation of the aorta, and acquired heart diseases such as rheumatic heart disease and bacterial endocarditis. It describes the pathophysiology, manifestations, and interventions for each condition.

Uploaded by

nieacatleya
Copyright
© Attribution Non-Commercial (BY-NC)
We take content rights seriously. If you suspect this is your content, claim it here.
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Download as DOCX, PDF, TXT or read online on Scribd
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Pediatric Nursing Reviewer

Cardiovascular Dysfunction
Fetal circulation:
Placenta - Umbilical vein – Liver (Ductus Venosus)
– Inferior Vena Cava – Right Atrium – Foramen
Ovale – Left Atrium – Mitral valve – left ventricle –
aortic semilunar valve – Aorta – ascending aorta –
head & upper extremities – superior vena cava –
right atrium – tricuspid valve – right ventricle –
pulmonary semilunar valve – pulmonary artery –
Lungs pulmonary vein – ductus arteriosus –
descending aorta – lower part of the body –
umbilical artery – placenta

INCREASE PULMONARY BLOOD FLOW


Atrial Septal Defect

CONGENITAL  In ASD Right Atrium Receives blood


INCIDENCE: both from the organs and from Left Atrium.
 5 – 8 in 1000 live birth.  In effect Right Ventricle and
 2 – 3 per 1000 birth is with Pulmonary Artery receives more blood than
symptoms needs treatment. they usually do.
 Major cause of death in first year of  PULMONARY CONGESTION is
life (after prematurity) common.
 Most common anomaly is VSD
Ventricular Septal Defect
Left to right shunting – Acyanotic

Increase Pulmonary blood flow

Congestive heart failure

Right to Left shunting – cyanotic

Decrease Pulmonary blood flow


 Presence of Hole between the Right
Hypoxemia Ventricle and the Left Ventricle.
 In VSD Right Ventricle Receives
Hemodynamics blood both from Right Atrium and from
1. Pressure Increase pressure in Left Side Left Ventricle.
Decrease pressure in Right Side  In effect Pulmonary Artery receives
2. Resistance Increase Resistance in Systemic more blood than they usually do.
Circulation Decrease in Pulmonary Circulation PULMONARY CONGESTION is common.
3. Saturation Severe cases:
SVC and IVC –lowest O2 saturation EISENMENGER SYNDROME – very severe
RA, RV and Pulmonary Artery - equal saturation resistance in pulmonary blood flow
Pulmonary Vein – fully saturated increases
LA and LV – equal saturation
Intervention in ASD and VSD
 If not interfere with the ADL – x
surgery
 If interfere with ADL – need for
surgery
 Put DACRON PATCH
 Open Heart Surgery is perform for
ASD and VSD

Patent Ductus Arteriosus


Manifestation
 The BP on the UPPER EXTREMITY is
GREATER relative to the pressure on the
LOWER EXTREMITY.
 Epistaxis
 Gum Bleeding
 Intracranial Hemorrhage – the most
common cause of death in COA.
Intervention
 Balloon – Tipped Catheter - to increase the
diameter of the lumen of the Aorta
 Presence of Artery that connects the  Usually done with Cardiac Catheterization.
Aorta to the Pulmonary Artery.
 If not effective – surgery is done by Ligating
 Shunting is from the Aorta to the portion of the Aorta with the coarctation and
Pulmonary Artery. then END-to-END Anastomosis is performed.
 In effect Pulmonary Artery receives
more blood that it usually does. CYANOTIC…
 Leading to increase Pulmonary DECREASE PULMONARY BLOOD FLOW
Artery Pressure.
 PULMONARY CONGESTION
Intervention of PDA
 15 mins to 12 hours (normal time it
takes for PDA to Close)
 After 12 hours - x surgery yet.
 INDOMETHACIN – prostaglandin
inhibitor that causes vasospasm of the Ductus
Arteriosus.
 Gastric irritant – causes Gastric
bleeding. Manifestation
Manifestaion of ASD & VSD  Clubbing of Fingers
 Presence of Murmur  Polycythemia
Manifestaion of PDA  TET SPELL / blue spell
 Presence of Murmur (machinery like Intervention
murmur)  Provide rest and Decrease Energy
expenditure.
OBSTRUCTIVE DEFECTS
 Position: Knee Chest
Coarctation of Aorta
Position, Squatting position.
COMPLETE REPAIR
 First yr of life
 Closure of VSD & resection of
stenosis; pericardial patch to enlarge RV
outflow
 Blalock –total repair

ACQUIRED HEART DISEASE


Rheumatic Heart Disease  Administration of Acetylsalicylic acid
 Inflammatory disease following an (ASA)
infection by GABHS.
Jones Criteria Bacterial Endocarditis
Major Criteria:  Infective endocarditis
 Subcutenous nodule  Infection of valve & inner lining of
 Polyarthritis heart that can damage & destroy heart valves
 Erythema marginatum  Usually affect mitral or aortic
 Carditis valve
 Syndenhamms Chorea or  After birth/ congenital heart defect
 St. Vitus Dance  Autoimmune; environmental factor;
Minor Criteria: infection
 Arthralgia  Sequela of bacteremia
 Low Grade Fever Manifestation
 All Lab results  Low grade fever, intermittent fever
**Increase C- Reactive Protein, ESR and  Headache, malaise, diaphoresis, wt
ASO loss
Diagnosis  New murmur – damage in
 2 MAJOR or valve/perforation
 1 MAJOR + 2 MINOR  Splenomegaly
Management  Petichiae
 CBR  Respi distress, dif, in feeding,
 Treatment of streptococcal tachycardia
tonsillitis/pharyngitis Intervention
 Medications = penicillin; ASA  High-dose antibiotics= penicillin IV
(tinnitus) (2-8wks)
Kawasaki Disease  DOC: Amoxicillin 1 hour before any
 Mucocutaneuos lymph node procedure
syndrome *dental prodedure; respi; GI; Gatrourinary
 Multisystem tract
disease associated with  Observe side effects of antibiotics; &
inflammation complications (embolism)
(Vasculitis)  Teaching importance of follow up
check up
Phases:  Early dx & tx
Acute Phase
- Fever Congestive heart failure
- Unresponsive to  Inability of the heart to pump
antibiotics & sufficiently to meet the metabolic needs of the
antipyretics body.
- Eyes redden, dry Common cause by congenital heart defect.
w/o drainage
- Strawberry
tongue Heart Failure
- Rashes Right Sided
Subacute Phase  Jugular vein distention
- 10 days after the onset.  Ascites
- Increase in Platelet count  Hepatomegaly
- Aneurysm
 Spleenomegaly
- Most dangerous phase
Convalescent Phase  Peripheral edema
th
- 25 – 40 days Left Sided
- ESR returning to normal  Dyspnea
Management  Orthopnea
 Crackles / Rales
 Moist cough  cardiac output & stroke volume
 Blood tinge frothy sputum Insertion of Cardiac Catheter
(Pulmonary Edema) 1. Right sided-(most common)
Intervention ** Femoral vein to right atrium
Digitalis – improves contractility. 2. Left sided
3 Major Actions **artery to aorta to Right ventricle
1. Increase force of contraction Management
2. Decrease heart rate  Consent
3. Enhances diuresis  sedation
Angiotensin Converting Enzyme (ACE)  Assess allergy to dye, seafood, or
inihbitors – it reduces afterload, thus make radiopaque dyes
heart easier to pump.  No solid food 6-8hours & liquid 4 hrs
Example:  Document ht. & wt.
Captopril (Capoten)
 VS; local anesthesia;
Enalapril (Vasotec)
 Check peripheral pulse
Diuretics - eliminate water and Salt
Example:  + fluttery feeling in insertion ; flushed;
Furosemide (Lasix) & Thiazides warm feeling when dye is injected; desired to
- It can cause K loss cough; palpitation = heart irritability
- K supplement  Shaving & cleaning the site
Decrease K = Enhancement of Digoxin that  IV Line
may lead to Digoxin Toxicity
Increase K = Decrease absorption of
Digoxin that may lead to no effect
Therefore normal K must be monitored
Normal K: 3.3-5.5mmol/L
Nursing Management
1. Administration of Digoxin
 Calculating correct dosage.
 Digoxin toxicity.
 Check . . .
APICAL PULSE
***Not Given if Pulse is:
a. < 90 – 110 beats/min – infant and
young children
b. 70 beats/min – older children
c. 60 beats / min adult
Digoxin Toxicity
 Bradycardia
 Anoxeria
 Nausea and Vomiting
Therapeutic Level: 0.8-2mcg/L
2. BP Monitoring
3. Position in SEMI FOLWERS - for Lung
expansion
4. Maintain Nutritional Needs – small frequent meal
5. Maintain F & Electrolyte Balance

Cardiac catheterization- most invasive


diagnostic procedure
 Inserting of catheter into the heart &
surrounding vessels
 Obtain info about structure &
 performance of the heart valves &
circulatory system; O2 sat.;
 pressure changes;

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