Papers by Michael Peterson
Journal of Cognitive Neuroscience, Oct 1, 2006
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Biological Psychiatry, Nov 1, 2011
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JAMA Psychiatry, Nov 1, 2013
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American Journal of Psychiatry, Feb 1, 2013
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BMC Psychiatry, Sep 18, 2012
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European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology, Jan 2, 2015
Benzodiazepines are commonly used medications that alter sleep spindles during non-rapid eye move... more Benzodiazepines are commonly used medications that alter sleep spindles during non-rapid eye movement (NREM) sleep, however the topographic changes to these functionally significant waveforms have yet to be fully elucidated. This study utilized high-density electroencephalography (hdEEG) to investigate topographic changes in sleep spindles and spindle-range activity caused by temazepam during NREM sleep in 18 healthy adults. After an accommodation night, sleep for all participants was recorded on two separate nights after taking either placebo or oral temazepam 15mg. Sleep was monitored using 256-channel hdEEG. Spectral analysis and spindle waveform detection of sleep EEG data were performed for each participant night. Global and topographic data were subsequently compared between temazepam and placebo conditions. Temazepam was associated with significant increases in spectral power from 10.33 to 13.83Hz. Within this frequency band, temazepam broadly increased sleep spindle duration...
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Sleep Medicine Clinics, 2015
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Psychiatry Research: Neuroimaging, 2012
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Journal of Affective Disorders, 2013
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Archives of General Psychiatry, 2012
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American Journal of Psychiatry, 2010
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MedEdPORTAL, 2020
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Clinical Neurophysiology, 2016
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Clinical Neurophysiology, 2016
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Archives of General Psychiatry, 2012
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WMJ : official publication of the State Medical Society of Wisconsin, 2018
Quality improvement (QI) education in residency training has become critical for numerous reasons... more Quality improvement (QI) education in residency training has become critical for numerous reasons, but little has been written about factors that lead to successful improvement projects within residency training. A quality improvement curriculum for third-year psychiatry residents was developed. The percentage of resident projects that have been successfully implemented was calculated. Residents completed the QI Knowledge Application Tool adapted for psychiatry before and after the curriculum to assess knowledge and skills. Eighteen of 19 resident projects were successfully implemented. QI Knowledge Application Tool scores improved from 4.8 to 8.1 ( = 0.0053) after completion of the curriculum. Residents are able to implement successful projects and to increase their knowledge and skills in quality improvement when given appropriate resources and incentives.
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Human brain mapping, Jan 19, 2015
Cognitive dysfunction is considered a core feature of schizophrenia, and impaired performances in... more Cognitive dysfunction is considered a core feature of schizophrenia, and impaired performances in episodic memory (EM) and executive function (EF) tasks are consistently reported in schizophrenia patients. Traditional fMRI and EEG studies have helped identifying brain areas, including the prefrontal cortex (PFC), involved in these tasks. However, it is unclear whether intrinsic defects in prefrontal function per se contribute to poor performance in schizophrenia, given the presence of confounds like reduced motivation and psychotic symptoms. TMS/hd-EEG measurements are obtained without cognitive effort, and can be calculated in any cortical area. We performed TMS/hd-EEG recordings in parietal, motor, premotor, and PFC in healthy individuals (N = 20) and schizophrenia patients (N = 20). Source modeling of TMS-evoked responses was performed, and measures of cortical activity (significant current density, SCD) and connectivity (significant current scattering, SCS) were computed. Patien...
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The Primary Care Companion For CNS Disorders, 2016
The incidence of posttraumatic stress disorder (PTSD) and obesity are on the rise, and evidence c... more The incidence of posttraumatic stress disorder (PTSD) and obesity are on the rise, and evidence continues to support the observation that individuals who have symptoms of PTSD are more likely to develop obesity in their lifetime. The incidence of obesity in individuals with PTSD, including war veterans, women, and children exposed to trauma, is not solely attributable to psychotropic medications, but actual pathophysiologic mechanisms have not been fully delineated. Additionally, there are no studies to date demonstrating that obese individuals are predisposed to developing PTSD compared to the general population. This review explores the pathogenic pathways common to both PTSD and obesity, which include inflammation, the renin-angiotensin-aldosterone system, cellular structures, and neuroendocrine activation. A PubMed search for the years 2000-2015 with the keywords PTSD and obesity was performed. There were no language restrictions. More research is needed in human subjects to understand the pathogenic pathways common to both PTSD and obesity and to further clarify the direction of identified associations. Ideally, in the future, clinical interventions targeting these pathways may be able to modify the course of PTSD and obesity. The outcome of studies investigating the utility of angiotensin-converting enzyme inhibitors and angiotensin receptor blockers in the treatment of PTSD symptoms will be relevant to control both PTSD and obesity. Importantly, outcomes assessing inflammation, obesity, and cardiac function in the same subjects also should be determined. Research is needed to reveal the multidimensional and intricate relationship between PTSD and obesity. The implications of this research would be essential for treatment, prevention, and potential public health reforms.
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Papers by Michael Peterson