Péter Lantos Hungarian doctor, neuropathologist, university professor
Lantos, Peter L., 1939-
Lantos, Péter 1939-
Lantos, Peter L.
VIAF ID: 115269400 (Personal)
Permalink: http://viaf.org/viaf/115269400
Preferred Forms
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- 100 1 _ ‡a Lantos, Peter L.
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- 100 1 _ ‡a Lantos, Peter L. (sparse)
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- 100 1 _ ‡a Lantos, Peter L., ‡d 1939-
- 100 1 _ ‡a Lantos, Peter ‡d 1939-
- 100 1 _ ‡a Lantos, Péter ‡d 1939-
- 100 0 _ ‡a Péter Lantos ‡c Hungarian doctor, neuropathologist, university professor
4xx's: Alternate Name Forms (19)
5xx's: Related Names (3)
- 551 _ _ ‡a London ‡4 ortw ‡4 https://d-nb.info/standards/elementset/gnd#placeOfActivity
- 551 _ _ ‡a Makó ‡g Csongrád ‡4 ortg ‡4 https://d-nb.info/standards/elementset/gnd#placeOfBirth
- 510 2 _ ‡a University of London ‡b Institute of Psychiatry ‡4 affi ‡4 https://d-nb.info/standards/elementset/gnd#affiliation ‡e Affiliation
Works
Title | Sources |
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Closed horizon, 2012: | |
Greenfield's neuropathology | |
Journey from childhood to Belsen | |
A medical research council randomized trial in patients with primary cerebral non-Hodgkin lymphoma: cerebral radiotherapy with and without cyclophosphamide, doxorubicin, vincristine, and prednisone chemotherapy | |
Neuronal density in the superior frontal and temporal gyri does not correlate with the degree of human immunodeficiency virus-associated dementia | |
Neuronal loss and neurofibrillary degeneration in the hippocampal cortex in late-onset sporadic Alzheimer's disease | |
Neuronal loss in symptom-free HIV infection | |
Neuronal number and volume alterations in the neocortex of HIV infected individuals. | |
Neuropathologic variation in frontotemporal dementia due to the intronic tau 10(+16) mutation | |
Neuropathological correlates of behavioural disturbance in confirmed Alzheimer's disease. | |
Neuropathology of early HIV-1 infection | |
New observation on ubiquitinated neurons in the cerebral cortex of multiple system atrophy (MSA) | |
A novel tau mutation (N296N) in familial dementia with swollen achromatic neurons and corticobasal inclusion bodies | |
Obituary of Mátyás Papp (1927-2019) | |
Office of Rare Diseases neuropathologic criteria for corticobasal degeneration. | |
Olfactory centres in Alzheimer's disease: olfactory bulb is involved in early Braak's stages. | |
Organic correlates of depressive symptoms in Alzheimer's dementia. Results of a prospective study, review of the literature | |
Papp-Lantos inclusions and the pathogenesis of multiple system atrophy: an update | |
Parallel lines | |
Patients with a novel neurofilamentopathy: dementia with neurofilament inclusions | |
Phenotypic variability in the brains of a family with a prion disease characterized by a 144-base pair insertion in the prion protein gene | |
Predominant deposition of amyloid-beta 42(43) in plaques in cases of Alzheimer's disease and hereditary cerebral hemorrhage associated with mutations in the amyloid precursor protein gene | |
Prion dementia without characteristic pathology | |
Prion protein immunocytochemistry helps to establish the true incidence of prion diseases | |
Prion protein immunocytochemistry--UK five centre consensus report | |
Progressive dendritic pathology in cynomolgus macaques infected with simian immunodeficiency virus | |
Progressive frontal gait disturbance with atypical Alzheimer's disease and corticobasal degeneration | |
Purkinje cell loss and astrocytosis in the cerebellum in familial and sporadic Alzheimer's disease | |
Quantification of pathological lesions in the frontal and temporal lobe of ten patients diagnosed with Pick's disease | |
Quantification of the pathological changes with laminar depth in the cortex in sporadic Creutzfeldt-Jakob disease | |
Quantitative analysis of tau isoform transcripts in sporadic tauopathies | |
A quantitative study of the pathological changes in white matter in multiple system atrophy | |
Recommended minimum data to be collected in research studies on Alzheimer's disease. The MRC (UK) Alzheimer's Disease Workshop Steering Committee | |
Relationships between beta-amyloid | |
The same prion strain causes vCJD and BSE | |
Sínek és sorsok | |
Size frequency distribution of prion protein | |
Spatial distribution of diffuse, primitive, and classic amyloid-beta deposits and blood vessels in the upper laminae of the frontal cortex in Alzheimer disease. | |
The spatial patterns of Lewy bodies, senile plaques, and neurofibrillary tangles in dementia with Lewy bodies | |
The spatial patterns of pathological brain lesions in 12 patients with corticobasal degeneration | |
The spatial patterns of prion protein deposits in cases of variant Creutzfeldt-Jakob disease | |
Spatial topography of the neurofibrillary tangles in cortical and subcortical regions in progressive supranuclear palsy | |
Species-barrier-independent prion replication in apparently resistant species | |
Sporadic and familial dementia with ubiquitin-positive tau-negative inclusions: clinical features of one histopathological abnormality underlying frontotemporal lobar degeneration | |
Structural and functional changes in skeletal muscle in anorexia nervosa. | |
Tau protein in the glial cytoplasmic inclusions of multiple system atrophy can be distinguished from abnormal tau in Alzheimer's disease. | |
Thalamic degeneration with negative prion protein immunostaining | |
Transmission of fatal familial insomnia to laboratory animals | |
Transport of thiamin across the blood-brain barrier of the rat in the absence of aerobic metabolism | |
Unaltered susceptibility to BSE in transgenic mice expressing human prion protein. | |
Validity and reliability of the preliminary NINDS neuropathologic criteria for progressive supranuclear palsy and related disorders. | |
Validity of current clinical criteria for Alzheimer's disease, vascular dementia and dementia with Lewy bodies. | |
The vasculature of experimental brain tumours. Part 4. The quantification of vascular permeability | |
Visual hallucinations are associated with lower alpha bungarotoxin binding in dementia with Lewy bodies | |
Von Ungarn nach Bergen-Belsen und zurück : eine Zeitreise | |
What can artificial neural networks teach us about neurodegenerative disorders with extrapyramidal features? | |
What determines the molecular composition of abnormal protein aggregates in neurodegenerative disease? | |
What does the study of the spatial patterns of pathological lesions tell us about the pathogenesis of neurodegenerative disorders? |