(Q47686942)

4 February 2018

review article

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title

Bcl-2-family proteins: the role of the BH3 domain in apoptosis (English)

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4 February 2018

main subject

BH4 domain binding

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Kelekar A

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4 February 2018

Thompson CB

series ordinal

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4 February 2018

publication date

1 August 1998

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4 February 2018

published in

Trends in Cell Biology

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4 February 2018

volume

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4 February 2018

issue

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4 February 2018

page(s)

324-330

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4 February 2018

The central executioners of apoptosis: caspases or mitochondria?

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7 January 2021

Cloning the chromosomal breakpoint of t(14;18) human lymphomas: clustering around JH on chromosome 14 and near a transcriptional unit on 18.

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Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death

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bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death

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7 January 2021

The E1B 19K protein blocks apoptosis by interacting with and inhibiting the p53-inducible and death-promoting Bax protein

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7 January 2021

Epstein-Barr virus-coded BHRF1 protein, a viral homologue of Bcl-2, protects human B cells from programmed cell death

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7 January 2021

C. elegans cell survival gene ced-9 encodes a functional homolog of the mammalian proto-oncogene bcl-2.

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7 January 2021

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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7 January 2021

Induction of apoptosis by the Bcl-2 homologue Bak

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7 January 2021

Bcl-x(S) anatagonizes the protective effects of Bcl-x(L).

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7 January 2021

Bok is a pro-apoptotic Bcl-2 protein with restricted expression in reproductive tissues and heterodimerizes with selective anti-apoptotic Bcl-2 family members

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7 January 2021

Mtd, a novel Bcl-2 family member activates apoptosis in the absence of heterodimerization with Bcl-2 and Bcl-XL

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7 January 2021

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

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7 January 2021

BID: a novel BH3 domain-only death agonist

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7 January 2021

Bim: a novel member of the Bcl-2 family that promotes apoptosis

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7 January 2021

harakiri, a novel regulator of cell death, encodes a protein that activates apoptosis and interacts selectively with survival-promoting proteins Bcl-2 and Bcl-X(L)

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7 January 2021

Blk, a BH3-containing mouse protein that interacts with Bcl-2 and Bcl-xL, is a potent death agonist

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7 January 2021

X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death

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7 January 2021

Identification of a novel regulatory domain in Bcl-X(L) and Bcl-2.

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Conversion of Bcl-2 to a Bax-like death effector by caspases

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Suppression of signalling through transcription factor NF-AT by interactions between calcineurin and Bcl-2.

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7 January 2021

The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4

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7 January 2021

Functional dissection of the human Bc12 protein: sequence requirements for inhibition of apoptosis

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7 January 2021

Bcl-2 functions in an antioxidant pathway to prevent apoptosis

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7 January 2021

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

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7 January 2021

Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c

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7 January 2021

Multiple Bcl-2 family members demonstrate selective dimerizations with Bax

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7 January 2021

BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

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7 January 2021

Structure of Bcl-xL-Bak peptide complex: recognition between regulators of apoptosis

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7 January 2021

The distal pathway of lipoprotein-induced cholesterol esterification, but not sphingomyelinase-induced cholesterol esterification, is energy-dependent.

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7 January 2021

Bcl-x(L) forms an ion channel in synthetic lipid membranes.

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7 January 2021

Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2.

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7 January 2021

Channel formation by antiapoptotic protein Bcl-2.

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7 January 2021

Inhibition of Bax channel-forming activity by Bcl-2

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7 January 2021

Mitochondrial control of nuclear apoptosis.

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7 January 2021

Mitochondrial permeability transition is a central coordinating event of apoptosis

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7 January 2021

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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7 January 2021

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

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7 January 2021

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

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7 January 2021

Bad is a BH3 domain-containing protein that forms an inactivating dimer with Bcl-XL.

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7 January 2021

Functional dissection of the pro-apoptotic protein Bik. Heterodimerization with anti-apoptosis proteins is insufficient for induction of cell death

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7 January 2021

Molecular Cloning of a Novel Polypeptide, DP5, Induced during Programmed Neuronal Death

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7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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7 January 2021

Interaction of CED-4 with CED-3 and CED-9: a molecular framework for cell death

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7 January 2021

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

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7 January 2021

Interaction and regulation of subcellular localization of CED-4 by CED-9

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7 January 2021

Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex

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7 January 2021

Regulation of apoptosis by BH3 domains in a cell-free system

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7 January 2021

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

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7 January 2021

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

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7 January 2021

10.1016/S0962-8924(98)01321-X

Identifiers

DOI

1 reference

4 February 2018

1 reference

4 February 2018