(Q45236710)

English

Level of beta-adrenergic receptor kinase 1 inhibition determines degree of cardiac dysfunction after chronic pressure overload-induced heart failure

scientific article published on 24 January 2005

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

11 December 2017

Level of beta-adrenergic receptor kinase 1 inhibition determines degree of cardiac dysfunction after chronic pressure overload-induced heart failure (English)

1 reference

Europe PubMed Central

PubMed publication ID

11 December 2017

1 reference

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Robert Lefkowitz

3

object named as

Robert J Lefkowitz

1 reference

Europe PubMed Central

PubMed publication ID

11 December 2017

author name string

Hideo Tachibana

1

1 reference

Europe PubMed Central

PubMed publication ID

11 December 2017

Sathyamangla V Naga Prasad

2

1 reference

Europe PubMed Central

PubMed publication ID

11 December 2017

Walter J Koch

4

1 reference

Europe PubMed Central

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11 December 2017

Howard A Rockman

5

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Europe PubMed Central

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11 December 2017

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publication date

24 January 2005

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Europe PubMed Central

PubMed publication ID

11 December 2017

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Europe PubMed Central

PubMed publication ID

11 December 2017

111

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Europe PubMed Central

PubMed publication ID

11 December 2017

5

1 reference

Europe PubMed Central

PubMed publication ID

11 December 2017

591-597

1 reference

Europe PubMed Central

PubMed publication ID

11 December 2017

Gbetagamma-dependent phosphoinositide 3-kinase activation in hearts with in vivo pressure overload hypertrophy

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Alterations in cardiac adrenergic signaling and calcium cycling differentially affect the progression of cardiomyopathy

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Expression of a beta-adrenergic receptor kinase 1 inhibitor prevents the development of myocardial failure in gene-targeted mice

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G protein-coupled receptor signalling in in vivo cardiac overload

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Hydraulic formula for calculation of the area of the stenotic mitral valve, other cardiac valves, and central circulatory shunts. I

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Expanding roles for beta-arrestins as scaffolds and adapters in GPCR signaling and trafficking.

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Germ-line transformation of mice

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Low- and high-level transgenic expression of beta2-adrenergic receptors differentially affect cardiac hypertrophy and function in Galphaq-overexpressing mice

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Decreased Catecholamine Sensitivity and β-Adrenergic-Receptor Density in Failing Human Hearts

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Seven-transmembrane-spanning receptors and heart function

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G-protein-coupled receptor kinase activity is increased in hypertension

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G protein-coupled receptors. III. New roles for receptor kinases and beta-arrestins in receptor signaling and desensitization

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Sensing heart stress

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Beta-adrenergic neuroeffector abnormalities in the failing human heart are produced by local rather than systemic mechanisms

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Inhibition of receptor-localized PI3K preserves cardiac beta-adrenergic receptor function and ameliorates pressure overload heart failure

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Beta-arrestin-dependent formation of beta2 adrenergic receptor-Src protein kinase complexes

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The beta(2)-adrenergic receptor mediates extracellular signal-regulated kinase activation via assembly of a multi-receptor complex with the epidermal growth factor receptor

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Caveolar localization dictates physiologic signaling of beta 2-adrenoceptors in neonatal cardiac myocytes

1 reference

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Phosphoinositide 3-kinase regulates beta2-adrenergic receptor endocytosis by AP-2 recruitment to the receptor/beta-arrestin complex

1 reference

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G protein-coupled receptor kinases

1 reference

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Cardiac function in mice overexpressing the beta-adrenergic receptor kinase or a beta ARK inhibitor

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Pleiotropy, homeostasis, and functional networks based on assays of cardiovascular traits in genetically randomized populations

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Agonist-dependent recruitment of phosphoinositide 3-kinase to the membrane by beta-adrenergic receptor kinase 1. A role in receptor sequestration

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G protein beta gamma subunits.

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Clathrin-coated vesicle formation and protein sorting: an integrated process

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Cardiac beta ARK1 inhibition prolongs survival and augments beta blocker therapy in a mouse model of severe heart failure

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Mechanism of beta-adrenergic receptor desensitization in cardiac hypertrophy is increased beta-adrenergic receptor kinase

1 reference

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Dual inhibition of beta-adrenergic and angiotensin II receptors by a single antagonist: a functional role for receptor-receptor interaction in vivo.

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Identifiers

10.1161/01.CIR.0000142291.70954.DF

1 reference

Europe PubMed Central

PubMed publication ID

11 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

11 December 2017

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