(Q41723645)

8 October 2017

title

The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant (English)

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8 October 2017

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Goldstein JC

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8 October 2017

Waterhouse NJ

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8 October 2017

Juin P

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8 October 2017

Evan GI

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8 October 2017

Green DR

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8 October 2017

language of work or name

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1 March 2000

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8 October 2017

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8 October 2017

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8 October 2017

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8 October 2017

page(s)

156-162

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Suicidal tendencies: apoptotic cell death by caspase family proteinases

8 October 2017

cites work

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7 January 2021

Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner

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7 January 2021

Caspases: killer proteases

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7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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7 January 2021

An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

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7 January 2021

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

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7 January 2021

Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development

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7 January 2021

Role of cytochrome c and dATP/ATP hydrolysis in Apaf-1-mediated caspase-9 activation and apoptosis

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7 January 2021

Autoactivation of procaspase-9 by Apaf-1-mediated oligomerization

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7 January 2021

Akt/Protein kinase B inhibits cell death by preventing the release of cytochrome c from mitochondria

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7 January 2021

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

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7 January 2021

Movement of Bax from the cytosol to mitochondria during apoptosis

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7 January 2021

Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xL.

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7 January 2021

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

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7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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7 January 2021

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

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7 January 2021

Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations

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7 January 2021

Mitochondrial permeability transition is a central coordinating event of apoptosis

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7 January 2021

Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells

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7 January 2021

Bcl-xL prevents cell death following growth factor withdrawal by facilitating mitochondrial ATP/ADP exchange

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7 January 2021

Mitochondrial release of caspase-2 and -9 during the apoptotic process

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7 January 2021

Molecular characterization of mitochondrial apoptosis-inducing factor

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7 January 2021

Mitochondria and apoptosis

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7 January 2021

The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins

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7 January 2021

Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

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7 January 2021

Simultaneous release of adenylate kinase and cytochrome c in cell death

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7 January 2021

Release of adenylate kinase 2 from the mitochondrial intermembrane space during apoptosis

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7 January 2021

The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol

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7 January 2021

Early redistribution of plasma membrane phosphatidylserine is a general feature of apoptosis regardless of the initiating stimulus: inhibition by overexpression of Bcl-2 and Abl

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7 January 2021

A novel assay for apoptosis. Flow cytometric detection of phosphatidylserine expression on early apoptotic cells using fluorescein labelled Annexin V

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7 January 2021

Bcl-xL functions downstream of caspase-8 to inhibit Fas- and tumor necrosis factor receptor 1-induced apoptosis of MCF7 breast carcinoma cells

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7 January 2021

The Mitochondrial F0F1-ATPase proton pump is required for function of the proapoptotic protein Bax in yeast and mammalian cells.

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7 January 2021

Cytochrome c and dATP-mediated oligomerization of Apaf-1 is a prerequisite for procaspase-9 activation

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7 January 2021

Intracellular adenosine triphosphate (ATP) concentration: a switch in the decision between apoptosis and necrosis

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7 January 2021

The mitochondrial death/life regulator in apoptosis and necrosis

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7 January 2021

Identifiers

DOI

10.1038/35004029

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8 October 2017

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