(Q40848403)

24 September 2017

title

Separation of cytochrome c-dependent caspase activation from thiol-disulfide redox change in cells lacking mitochondrial DNA. (English)

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24 September 2017

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24 September 2017

Cai J

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24 September 2017

Wallace DC

series ordinal

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24 September 2017

Jones DP

series ordinal

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24 September 2017

publication date

1 August 2000

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Free Radical Biology and Medicine

24 September 2017

published in

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24 September 2017

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24 September 2017

issue

3-4

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24 September 2017

page(s)

334-342

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Oxygen radicals and signaling

24 September 2017

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Caspases: enemies within

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Rapid and specific reactive oxygen species generation via NADPH oxidase activation during Fas-mediated apoptosis

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Prevention of mitochondrial injury by manganese superoxide dismutase reveals a primary mechanism for alkaline-induced cell death

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Thioredoxin peroxidase is a novel inhibitor of apoptosis with a mechanism distinct from that of Bcl-2

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N-acetyl-L-cysteine is a pluripotent protector against cell death and enhancer of trophic factor-mediated cell survival in vitro

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Chronic ethanol ingestion impairs alveolar type II cell glutathione homeostasis and function and predisposes to endotoxin-mediated acute edematous lung injury in rats

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7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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7 January 2021

Importance of the redox state of cytochrome c during caspase activation in cytosolic extracts

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Rapid and specific efflux of reduced glutathione during apoptosis induced by anti-Fas/APO-1 antibody

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Glutathione redox potential in response to differentiation and enzyme inducers.

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Marked increase in the number and variety of mitochondrial DNA rearrangements in aging human skeletal muscle

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Mitochondrial DNA damage is more extensive and persists longer than nuclear DNA damage in human cells following oxidative stress

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7 January 2021

REDUCTION-POTENTIAL OF GLUTATHIONE

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7 January 2021

Mitochondria and apoptosis

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7 January 2021

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

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7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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7 January 2021

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7 January 2021

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7 January 2021

Presence of a pre-apoptotic complex of pro-caspase-3, Hsp60 and Hsp10 in the mitochondrial fraction of jurkat cells

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

The caspase-3 precursor has a cytosolic and mitochondrial distribution: implications for apoptotic signaling

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7 January 2021

Cytochrome c-mediated apoptosis in cells lacking mitochondrial DNA. Signaling pathway involving release and caspase 3 activation is conserved

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7 January 2021

10.1016/S0891-5849(00)00312-9

Identifiers

DOI

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24 September 2017

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