(Q40603687)

22 September 2017

title

A key role for TRPM7 channels in anoxic neuronal death (English)

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22 September 2017

Michelle Aarts

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22 September 2017

Koji Iihara

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22 September 2017

Wen-Li Wei

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22 September 2017

Zhi-Gang Xiong

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22 September 2017

Mark Arundine

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22 September 2017

Waldy Cerwinski

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22 September 2017

John F MacDonald

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22 September 2017

Michael Tymianski

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22 September 2017

language of work or name

English

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publication date

1 December 2003

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22 September 2017

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22 September 2017

volume

115

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22 September 2017

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22 September 2017

page(s)

863-877

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Treatment of ischemic brain damage by perturbing NMDA receptor- PSD-95 protein interactions

22 September 2017

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RNA interference: antiviral defense and genetic tool

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Mitochondrial production of reactive oxygen species in cortical neurons following exposure to N-methyl-D-aspartate

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Duplexes of 21-nucleotide RNAs mediate RNA interference in cultured mammalian cells

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Prolonged enhancement and depression of synaptic transmission in CA1 pyramidal neurons induced by transient forebrain ischemia in vivo

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7 January 2021

Combined oxygen and glucose deprivation in cortical cell culture: calcium-dependent and calcium-independent mechanisms of neuronal injury

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Spatially and functionally distinct Ca2+ stores in sarcoplasmic and endoplasmic reticulum

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7 January 2021

LTRPC2 Ca2+-permeable channel activated by changes in redox status confers susceptibility to cell death

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Why did NMDA receptor antagonists fail clinical trials for stroke and traumatic brain injury?

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7 January 2021

Polyvalent cations as permeant probes of MIC and TRPM7 pores

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MIC channels are inhibited by internal divalent cations but not ATP.

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TRPM7 provides an ion channel mechanism for cellular entry of trace metal ions

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7 January 2021

The TRP channels, a remarkably functional family

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LTRPC7 is a Mg.ATP-regulated divalent cation channel required for cell viability

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Brain lesions, obesity, and other disturbances in mice treated with monosodium glutamate

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Requirement for superoxide in excitotoxic cell death

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TRPM2 Ca2+ permeable cation channels: from gene to biological function

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Glutamate release in severe brain ischaemia is mainly by reversed uptake.

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7 January 2021

TRP-PLIK, a bifunctional protein with kinase and ion channel activities

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7 January 2021

The TRPM7 channel is inactivated by PIP(2) hydrolysis

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7 January 2021

Determination of the time course and extent of neurotoxicity at defined temperatures in cultured neurons using a modified multiwell plate fluorescence scanner

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7 January 2021

Distinct influx pathways, not calcium load, determine neuronal vulnerability to calcium neurotoxicity.

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7 January 2021

Specific coupling of NMDA receptor activation to nitric oxide neurotoxicity by PSD-95 protein

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Source specificity of early calcium neurotoxicity in cultured embryonic spinal neurons

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7 January 2021

Activation of the cation channel long transient receptor potential channel 2 (LTRPC2) by hydrogen peroxide. A splice variant reveals a mode of activation independent of ADP-ribose

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7 January 2021

AMPA receptor activation potentiates zinc neurotoxicity

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7 January 2021

Extracellular calcium sensed by a novel cation channel in hippocampal neurons

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7 January 2021

Differing effects of copper,zinc superoxide dismutase overexpression on neurotoxicity elicited by nitric oxide, reactive oxygen species, and excitotoxins

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7 January 2021

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Identifiers

DOI

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22 September 2017

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