(Q28567073)

31 January 2017

title

Dominant-negative c-Jun promotes neuronal survival by reducing BIM expression and inhibiting mitochondrial cytochrome c release (English)

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31 January 2017

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J. Whitfield

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31 January 2017

S. J. Neame

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31 January 2017

L. Paquet

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31 January 2017

O. Bernard

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31 January 2017

J. Ham

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31 January 2017

language of work or name

English

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publication date

1 March 2001

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31 January 2017

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31 January 2017

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31 January 2017

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31 January 2017

page(s)

629–643

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p53 is essential for developmental neuron death as regulated by the TrkA and p75 neurotrophin receptors

31 January 2017

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Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

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7 January 2021

Temporal analysis of events associated with programmed cell death (apoptosis) of sympathetic neurons deprived of nerve growth factor

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7 January 2021

BAX is required for neuronal death after trophic factor deprivation and during development

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7 January 2021

Placement of the BCL2 family member BAX in the death pathway of sympathetic neurons activated by trophic factor deprivation.

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Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

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Evidence of a novel event during neuronal death: development of competence-to-die in response to cytoplasmic cytochrome c

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Genetic and metabolic status of NGF-deprived sympathetic neurons saved by an inhibitor of ICE family proteases

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

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Characterization of apoptosis in cultured rat sympathetic neurons after nerve growth factor withdrawal

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Stress-induced Fas ligand expression in T cells is mediated through a MEK kinase 1-regulated response element in the Fas ligand promoter

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bcl-2 transgene expression can protect neurons against developmental and induced cell death

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7 January 2021

Bax directly induces release of cytochrome c from isolated mitochondria

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7 January 2021

A simple and efficient method for purification of infectious recombinant adenovirus.

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7 January 2021

DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

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7 January 2021

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7 January 2021

Withdrawal of survival factors results in activation of the JNK pathway in neuronal cells leading to Fas ligand induction and cell death

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7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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7 January 2021

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7 January 2021

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7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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7 January 2021

Inhibitors of protein synthesis and RNA synthesis prevent neuronal death caused by nerve growth factor deprivation

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7 January 2021

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

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7 January 2021

Blocking cytochrome c activity within intact neurons inhibits apoptosis.

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7 January 2021

Bim: a novel member of the Bcl-2 family that promotes apoptosis

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7 January 2021

An Essential Role for Rho, Rac, and Cdc42 GTPases in Cell Cycle Progression Through G 1

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex

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7 January 2021

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7 January 2021

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7 January 2021

p53 induces apoptosis by caspase activation through mitochondrial cytochrome c release.

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7 January 2021

Apoptosis in neurodegenerative disorders

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7 January 2021

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7 January 2021

Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

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7 January 2021

BID: a novel BH3 domain-only death agonist

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7 January 2021

Phosphorylation of c-Jun is necessary for apoptosis induced by survival signal withdrawal in cerebellar granule neurons.

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7 January 2021

The anti-apoptotic protein ITA is essential for NGF-mediated survival of embryonic chick neurons

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7 January 2021

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

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https://api.crossref.org/works/10.1016%2FS0896-6273%2801%2900239-2

7 January 2021

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

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https://api.crossref.org/works/10.1016%2FS0896-6273%2801%2900239-2

7 January 2021

Apoptosis in the nervous system

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https://api.crossref.org/works/10.1016%2FS0896-6273%2801%2900239-2

7 January 2021