(Q24554454)

Revision as of 19:16, 29 January 2017 by Harej (talk | contribs) (‎Created claim: cites work (P2860): Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L) (Q28297419), #quickstatements)

(diff) ← Older revision | Latest revision (diff) | Newer revision → (diff)

English

NF-kappaB induces expression of the Bcl-2 homologue A1/Bfl-1 to preferentially suppress chemotherapy-induced apoptosis

scientific article

In more languages

default for all languages

No label defined

No description defined

Statements

instance of

scholarly article

0 references

title

NF-kappaB induces expression of the Bcl-2 homologue A1/Bfl-1 to preferentially suppress chemotherapy-induced apoptosis (English)

0 references

0 references

C Y Wang

0 references

D C Guttridge

series ordinal

0 references

M W Mayo

series ordinal

0 references

A S Baldwin

series ordinal

0 references

original language of film or TV show

English

0 references

publication date

September 1999

0 references

published in

Molecular and Cellular Biology

0 references

volume

0 references

issue

0 references

page(s)

5923-9

0 references

cites work

The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappaB that blocks TNFalpha-induced apoptosis

1 reference

stated in

PubMed Central

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

stated in

PubMed Central

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

stated in

PubMed Central

IEX-1L, an apoptosis inhibitor involved in NF-kappaB-mediated cell survival

1 reference

stated in

PubMed Central

IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspases

1 reference

stated in

PubMed Central

Bcl-XL interacts with Apaf-1 and inhibits Apaf-1-dependent caspase-9 activation

1 reference

stated in

PubMed Central

Human IAP-like protein regulates programmed cell death downstream of Bcl-xL and cytochrome c

1 reference

stated in

PubMed Central

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

stated in

PubMed Central

The c-IAP-1 and c-IAP-2 proteins are direct inhibitors of specific caspases

1 reference

stated in

PubMed Central

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

1 reference

stated in

PubMed Central

The central executioner of apoptosis: multiple connections between protease activation and mitochondria in Fas/APO-1/CD95- and ceramide-induced apoptosis

1 reference

stated in

PubMed Central

DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis

1 reference

stated in

PubMed Central

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

1 reference

stated in

PubMed Central

Bcl-2 targets the protein kinase Raf-1 to mitochondria

1 reference

stated in

PubMed Central

The NF-kappa B and I kappa B proteins: new discoveries and insights

1 reference

stated in

PubMed Central

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

1 reference

stated in

PubMed Central

A conserved family of cellular genes related to the baculovirus iap gene and encoding apoptosis inhibitors

1 reference

stated in

PubMed Central

NF-kappa B and Rel proteins: evolutionarily conserved mediators of immune responses

1 reference

stated in

PubMed Central

Cloning of human Bcl-2 homologue: inflammatory cytokines induce human A1 in cultured endothelial cells

1 reference

stated in

PubMed Central

IAP family proteins--suppressors of apoptosis

1 reference

stated in

PubMed Central

Modulation of life and death by the TNF receptor superfamily

1 reference

stated in

PubMed Central

Death receptors: signaling and modulation

1 reference

stated in

PubMed Central

Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes

1 reference

stated in

PubMed Central

Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex

1 reference

stated in

PubMed Central

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

0 references

Identifiers

DOI

10.1128/MCB.19.9.5923

0 references

PMC publication ID

84448

0 references

PubMed publication ID

10454539

0 references

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)

Retrieved from "https://www.wikidata.org/w/index.php?title=Q24554454&oldid=438832493"